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These are based on the refinement of the immune model of infectious disease developed by \\textit{M. A. Nowak} and \\textit{C. R. Bangham} [Science 272, 74-79 (1996)] which does not assume interaction between different types of CTLs.   The authors investigate the stability of equilibria for the one-virus model and proceed with numerical simulations for the two-virus model. It turns out that the frequency dependence caused by the random search with viral diversity leads to continuous changes of dominant infected cells and corresponding specific immune cells which, in turn, yield complex behavior. In particular, it has been demonstrated that the frequency dependence may lead to the loss of stability of the interior equilibrium of the one-virus model. On the other hand, numerical simulations suggest that only a stable limit cycle exists when the interior equilibrium is unstable. 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