Mathematical oncology (Q1648952): Difference between revisions

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+https://doi.org/10.1007/s11538-018-0423-5
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+W4244283019
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+Q88427756
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+Size matters: metastatic cluster size and stromal recruitment in the establishment of successful prostate cancer to bone metastases
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+Stability analysis of a model of interaction between the immune system and cancer cells in chronic myelogenous leukemia
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+A poroelasticity theory approach to study the mechanisms leading to elevated interstitial fluid pressure in solid tumours
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+The role of exosomes in pancreatic cancer microenvironment
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+How nucleus mechanics and ECM microstructure influence the invasion of single cells and multicellular aggregates
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+Adaptation to stochastic temporal variations in intratumoral blood flow: the Warburg effect as a bet hedging strategy
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+Models for the Growth of a Solid Tumor by Diffusion
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+Model prediction and validation of an order mechanism controlling the spatiotemporal phenotype of early hepatocellular carcinoma
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+Mathematical modeling of cellular cross-talk between endothelial and tumor cells highlights counterintuitive effects of VEGF-targeted therapies
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+Stability of control networks in autonomous homeostatic regulation of stem cell lineages
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+The evolution of tumour composition during fractionated radiotherapy: implications for outcome
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+Simulating PDGF-driven glioma growth and invasion in an anatomically accurate brain domain
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+Predicting patient-specific radiotherapy protocols based on mathematical model choice for proliferation saturation index
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+A patient-specific anisotropic diffusion model for brain tumour spread
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+Computational modelling of cancer development and growth: modelling at multiple scales and multiscale modelling
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+Ordinary differential equation models for adoptive immunotherapy
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+Additive damage models for cellular pharmacodynamics of radiation-chemotherapy combinations
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+Inferring intracellular signal transduction circuitry from molecular perturbation experiments
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+Three-dimensional spatiotemporal modeling of colon cancer organoids reveals that multimodal control of stem cell self-renewal is a critical determinant of size and shape in early stages of tumor growth
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