Analysis of a nonlinear age-structured tumor cell population model (Q2296871): Difference between revisions

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-LeiNa Zhao
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-Normal rank
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+LeiNa Zhao
@@ Property / author: LeiNa Zhao / rank @@
+Normal rank
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+MaRDI publication profile
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+Normal rank
@@ Property / full work available at URL @@
+https://doi.org/10.1007/s11071-019-05190-4
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+W2968456704
@@ Property / OpenAlex ID: W2968456704 / rank @@
+Normal rank
@@ Property / cites work @@
+Steady states and outbreaks of two-phase nonlinear age-structured model of population dynamics with discrete time delay
+Normal rank
@@ Property / cites work @@
+Nutrient limitations as an explanation of Gompertzian tumor growth
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+A history of the study of solid tumour growth: the contribution of mathematical modelling
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+Asymptotic Analysis of a Cell Cycle Model Based on Unequal Division
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+Necessary and sufficient conditions for asynchronous exponential growth in age structured cell populations with quiescence
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+Computational Methods and Results for Structured Multiscale Models of Tumor Invasion
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+An age-and-cyclin-structured cell population model for healthy and tumoral tissues
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+Global Behavior of an Age-Structured Epidemic Model
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+Asynchronous exponential growth in an age structured population of proliferating and quiescent cells
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+The contribution of age structure to cell population responses to targeted therapeutics
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+Non-linear age-dependent population dynamics
@@ Property / cites work: Non-linear age-dependent population dynamics / rank @@
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@@ Property / cites work @@
+Age-size structure in populations with quiescence
@@ Property / cites work: Age-size structure in populations with quiescence / rank @@
+Normal rank
@@ Property / cites work @@
+Asynchronous exponential growth of semigroups of nonlinear operators
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@@ Property / cites work @@
+Mathematical analysis of an age-structured SIR epidemic model with vertical transmission
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@@ Property / cites work @@
+Q3461790
@@ Property / cites work: Q3461790 / rank @@
+Normal rank
@@ Property / cites work @@
+Modelling the balance between quiescence and cell death in normal and tumour cell populations
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