Mathematical analysis of two competing cancer cell migration mechanisms driven by interstitial fluid flow (Q2190704): Difference between revisions

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+Dynamic theory of quasilinear parabolic systems. III: Global existence
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+Global classical small-data solutions for a three-dimensional chemotaxis Navier-Stokes system involving matrix-valued sensitivities
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+Global Existence and Temporal Decay in Keller-Segel Models Coupled to Fluid Equations
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+Global Solutions to the Coupled Chemotaxis-Fluid Equations
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+A Stokes Two-Fluid Model for Cell Migration that Can Account for Physical Cues in the Microenvironment
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+Stabilization in a chemotaxis model for tumor invasion
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+Suppressing chemotactic blow-up through a fast splitting scenario on the plane
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+Boundedness vs. blow-up in a chemotaxis system
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+Biomixing by Chemotaxis and Enhancement of Biological Reactions
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+Suppression of chemotactic explosion by mixing
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+Existence and uniqueness theorem on mild solutions to the Keller-Segel system coupled with the Navier-Stokes fluid
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+Q5562267
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+Long-term behaviour in a chemotaxis-fluid system with logistic source
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+Global small-data solutions of a two-dimensional chemotaxis system with rotational flux terms
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+Hölder estimates for local solutions of some doubly nonlinear degenerate parabolic equations
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+Bacterial swimming and oxygen transport near contact lines
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+Global Large-Data Solutions in a Chemotaxis-(Navier–)Stokes System Modeling Cellular Swimming in Fluid Drops
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+Stabilization in a two-dimensional chemotaxis-Navier-Stokes system
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+Global weak solutions in a three-dimensional chemotaxis-Navier-Stokes system
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+How far do chemotaxis-driven forces influence regularity in the Navier-Stokes system?
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+Can Rotational Fluxes Impede the Tendency Toward Spatial Homogeneity in Nutrient Taxis(-Stokes) Systems?
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