Pages that link to "Item:Q2655400"
From MaRDI portal
The following pages link to Electromechanics of the heart: a unified approach to the strongly coupled excitation-contraction problem (Q2655400):
Displaying 50 items.
- Atrial and ventricular fibrillation: computational simulation of spiral waves in cardiac tissue (Q363104) (← links)
- A fully implicit finite element method for bidomain models of cardiac electromechanics (Q465807) (← links)
- Stability of active muscle tissue (Q525440) (← links)
- A generic approach towards finite growth with examples of athlete's heart, cardiac dilation, and cardiac wall thickening (Q602803) (← links)
- Computational modeling of electrochemical coupling: a novel finite element approach towards ionic models for cardiac electrophysiology (Q660312) (← links)
- Primal-mixed formulations for reaction-diffusion systems on deforming domains (Q729280) (← links)
- The generalized Hill model: a kinematic approach towards active muscle contraction (Q904802) (← links)
- New sets of spectral invariants for electro-elastic bodies with one and two families of fibres (Q1658084) (← links)
- The living heart project: a robust and integrative simulator for human heart function (Q1669467) (← links)
- Computational modeling of coupled cardiac electromechanics incorporating cardiac dysfunctions (Q1669471) (← links)
- Thermodynamically consistent orthotropic activation model capturing ventricular systolic wall thickening in cardiac electromechanics (Q1669482) (← links)
- A multiscale model for eccentric and concentric cardiac growth through sarcomerogenesis (Q1720100) (← links)
- A fully coupled model for electromechanics of the heart (Q1929630) (← links)
- Active stress vs. active strain in mechanobiology: constitutive issues (Q1937176) (← links)
- Machine learning in drug development: characterizing the effect of 30 drugs on the QT interval using Gaussian process regression, sensitivity analysis, and uncertainty quantification (Q1987901) (← links)
- A new computational framework for electro-activation in cardiac mechanics (Q1987938) (← links)
- Non-conforming finite-element formulation for cardiac electrophysiology: an effective approach to reduce the computation time of heart simulations without compromising accuracy (Q1990715) (← links)
- A highly parallel implicit domain decomposition method for the simulation of the left ventricle on unstructured meshes (Q1995006) (← links)
- Modeling cardiac muscle fibers in ventricular and atrial electrophysiology simulations (Q2020750) (← links)
- A comparative study of fully implicit staggered and monolithic solution methods. I: Coupled bidomain equations of cardiac electrophysiology (Q2075955) (← links)
- Cardiac electro-mechanical activity in a deforming human cardiac tissue: modeling, existence-uniqueness, finite element computation and application to multiple ischemic disease (Q2113516) (← links)
- How viscous is the beating heart? Insights from a computational study (Q2171510) (← links)
- Towards an efficient computational strategy for electro-activation in cardiac mechanics (Q2173633) (← links)
- New classes of electro-elastic and thermo-electro-elastic bodies that are not Green elastic (Q2183822) (← links)
- An intergrid transfer operator using radial basis functions with application to cardiac electromechanics (Q2205162) (← links)
- (INVITED) Reaction-diffusion waves in cardiovascular diseases (Q2223373) (← links)
- A curvilinear isogeometric framework for the electromechanical activation of thin muscular tissues (Q2237275) (← links)
- Modeling the chemoelectromechanical behavior of skeletal muscle using the parallel open-source software library OpenCMISS (Q2262228) (← links)
- Fluid-structure-electrophysiology interaction (FSEI) in the left-heart: a multi-way coupled computational model (Q2282212) (← links)
- Computational cardiology: a modified Hill model to describe the electro-visco-elasticity of the myocardium (Q2308913) (← links)
- The importance of mechano-electrical feedback and inertia in cardiac electromechanics (Q2309848) (← links)
- A matrix DEIM technique for model reduction of nonlinear parametrized problems in cardiac mechanics (Q2309983) (← links)
- A viscoactive constitutive modeling framework with variational updates for the myocardium (Q2310357) (← links)
- Patient-specific modeling for left ventricular mechanics using data-driven boundary energies (Q2310369) (← links)
- Parallel multilevel solvers for the cardiac electro-mechanical coupling (Q2349311) (← links)
- Anatomically accurate high resolution modeling of human whole heart electromechanics: A strongly scalable algebraic multigrid solver method for nonlinear deformation (Q2374953) (← links)
- Interoperable executive library for the simulation of biomedical processes (Q2517440) (← links)
- Newton-Krylov-BDDC solvers for nonlinear cardiac mechanics (Q2631461) (← links)
- 3D-0D closed-loop model for the simulation of cardiac biventricular electromechanics (Q2670385) (← links)
- Bioelectrical effects of mechanical feedbacks in a strongly coupled cardiac electro-mechanical model (Q2788505) (← links)
- An active strain electromechanical model for cardiac tissue (Q2900422) (← links)
- Coupled electromechanical model of the heart: Parallel finite element formulation (Q2900424) (← links)
- A model and simulation of uterine contractions (Q2950710) (← links)
- Computational modeling of passive myocardium (Q3084132) (← links)
- Segregated Algorithms for the Numerical Simulation of Cardiac Electromechanics in the Left Human Ventricle (Q3300479) (← links)
- Mathematical analysis and 2-scale convergence of a heterogeneous microscopic bidomain model (Q4568508) (← links)
- The cardiovascular system: Mathematical modelling, numerical algorithms and clinical applications (Q4594245) (← links)
- Influence of myocardial fiber/sheet orientations on left ventricular mechanical contraction (Q5137408) (← links)
- Solvability analysis and numerical approximation of linearized cardiac electromechanics (Q5247101) (← links)
- Modeling and simulation of human induced pluripotent stem cell‐derived cardiac tissue (Q6067372) (← links)