Modeling fibrous cap formation in atherosclerotic plaque development: stability and oscillatory behavior

DOI10.1186/S13662-017-1252-9zbMATH Open1422.92076OpenAlexW2735688697WikidataQ59611887 ScholiaQ59611887MaRDI QIDQ1631954FDOQ1631954

Authors: Wanwarat Anlamlert, Y. Lenbury, Jonathan Bell 

Publication date: 7 December 2018

Published in: Advances in Difference Equations (Search for Journal in Brave)

Full work available at URL: https://doi.org/10.1186/s13662-017-1252-9

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zbMATH Keywords

atherosclerosisHopf bifurcationsystem stabilityatherosclerotic plaque growth

Mathematics Subject Classification ID

Medical applications (general) (92C50) Dynamical systems in biology (37N25)

Cites Work

• Mathematical and numerical modeling of early atherosclerotic lesions
• Title not available (Why is that?)
• Long time evolution of atherosclerotic plaques
• Growth of necrotic cores in atherosclerotic plaque
• Stability analysis of a reaction-diffusion system modeling atherogenesis

Cited In (20)

• Frontiers in mathematical modelling of the lipid metabolism under normal conditions and its alterations in heart diseases
• Mathematical modeling and simulation of the evolution of plaques in blood vessels
• A multiphase model of growth factor-regulated atherosclerotic cap formation
• An ODE model of early stages of atherosclerosis: mechanisms of the inflammatory response
• Mathematical modeling of atherosclerotic plaque destabilization: role of neovascularization and intraplaque hemorrhage
• Stability analysis of a model of atherosclerotic plaque growth
• Mathematical modelling of the effects of statins on the growth of necrotic core in atherosclerotic plaque
• Mathematical analysis and numerical simulations for a model of atherosclerosis
• Studies on atherosclerotic plaque formation: a mathematical approach
• Stability analysis of mathematical modeling of atherosclerotic plaque formation
• Bifurcation for a free boundary model of an atherosclerotic plaque formation in the presence of reverse cholesterol transport with Holling type III functional response
• A new lipid-structured model to investigate the opposing effects of LDL and HDL on atherosclerotic plaque macrophages
• Bifurcation and dynamics in a mathematical model of early atherosclerosis. How acute inflammation drives lesion development
• Foam cell formation in atherosclerosis: HDL and macrophage reverse cholesterol transport
• Mathematical modeling of inflammatory processes
• Atherosclerosis: the carbonic anhydrase, carbon dioxide, calcium concerted theory
• Modelling preferential phagocytosis in atherosclerosis: delineating timescales in plaque development
• Mathematical model of hypertension-induced arterial remodeling: a chemo-mechanical approach
• A lipid-structured model of atherosclerotic plaque macrophages with lipid-dependent kinetics
• A two-phase model of early fibrous cap formation in atherosclerosis

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