A biophysical model of the cortex-basal ganglia-thalamus network in the 6-OHDA lesioned rat model of Parkinson's disease
DOI10.1007/S10827-016-0593-9zbMATH Open1382.92065DBLPjournals/jcns/KumaraveluBG16OpenAlexW2296941957WikidataQ37156622 ScholiaQ37156622MaRDI QIDQ1704973FDOQ1704973
Authors: Karthik Kumaravelu, David T. Brocker, Warren M. Grill 
Publication date: 14 March 2018
Published in: Journal of Computational Neuroscience (Search for Journal in Brave)
Full work available at URL: http://europepmc.org/articles/pmc4975943
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Cites Work
Cited In (13)
- Mean-field modeling of the basal ganglia-thalamocortical system. I: Firing rates in healthy and Parkinsonian states
- Optogenetic stimulation of primary motor cortex regulates beta oscillations in the basal ganglia: a computational study
- Toward a cerebello-thalamo-cortical computational model of spinocerebellar ataxia
- BLENX MODELS OF α-SYNUCLEIN AND PARKIN KINETICS IN NEUROPATHOLOGY OF PARKINSON'S DISEASE
- The possible mechanism of direct feedback projections from basal ganglia to cortex in beta oscillations of Parkinson's disease: a theoretical evidence in the competing resonance model
- Deep brain stimulation for movement disorder treatment: exploring frequency-dependent efficacy in a computational network model
- Hopf bifurcation analysis for Parkinson oscillation with heterogeneous delays: a theoretical derivation and simulation analysis
- Multiple-site deep brain stimulation with delayed rectangular waveforms for Parkinson's disease
- The effects of DBS patterns on basal ganglia activity and thalamic relay
- Control analysis of optogenetics and deep brain stimulation targeting basal ganglia for Parkinson's disease
- Firing activities and magnetic stimulation effects in a cortico-basal ganglia-thalamus neural network
- Relative contributions of local cell and passing fiber activation and silencing to changes in thalamic fidelity during deep brain stimulation and lesioning: a computational modeling study
- Origins and suppression of oscillations in a computational model of Parkinson's disease
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