Mathematical analysis of the global dynamics of a HTLV-I infection model, considering the role of cytotoxic T-lymphocytes
DOI10.1016/J.MATCOM.2020.09.009OpenAlexW3086654508MaRDI QIDQ1998277FDOQ1998277
Authors: Subhas Khajanchi, Sovan Bera, Tapan Kumar Roy
Publication date: 6 March 2021
Published in: Mathematics and Computers in Simulation (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1016/j.matcom.2020.09.009
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Genetics and population dynamics (92Dxx) General theory of linear operators (47Axx) Physiological, cellular and medical topics (92Cxx) General first-order partial differential equations and systems of first-order partial differential equations (35Fxx)
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Cited In (33)
- A co-infection model on TB - COVID-19 with optimal control and sensitivity analysis
- Mathematical modeling of COVID‐19 and Omicron outbreak spread: Optimal control approach for intervention strategies
- Population dynamics of HTLV-I infection: A discrete-time mathematical epidemic model approach
- Non-fractional and fractional mathematical analysis and simulations for Q fever
- Dynamics and optimal therapy of a stochastic HTLV-1 model incorporating Ornstein-Uhlenbeck process
- Change time estimation uncertainty in nonlinear dynamical systems with applications to COVID‐19
- A Fractional Order SITR Model for Forecasting of Transmission of COVID-19: Sensitivity Statistical Analysis
- Modeling the dynamical behavior of the interaction of T-cells and human immunodeficiency virus with saturated incidence
- Stability and optimal control of human T-cell lymphotropic virus type I (HTLV-I) infection of CD4+ T-cells which leads to leukemia model with prevention and treatment
- A time scale approach for analyzing pathogenesis of ATL development associated with HTLV-1 infection
- The impact of multiple population-wide testing and social distancing on the transmission of an infectious disease
- Modelling the potential impact of social distancing on the COVID-19 epidemic in South Africa
- A reaction-diffusion epidemic model with virus mutation and media coverage: theoretical analysis and numerical simulation
- The COVID-19 pandemic as inspiration to reconsider epidemic models: a novel approach to spatially homogeneous epidemic spread modeling
- Stochastic persistence, extinction and stationary distribution in HTLV-I infection model with CTL immune response
- Correcting notification delay and forecasting of COVID-19 data
- Modelling the impact of media-induced social distancing on the containment of COVID-19 in Beijing
- Threshold dynamics and probability density function of a stochastic multi-strain coinfection model with amplification and vaccination
- Unequal effects of SARS-CoV-2 infections: model of SARS-CoV-2 dynamics in Cameroon (Sub-Saharan Africa) versus New York State (United States)
- Dynamical analysis of a nonlocal delayed and diffusive HIV latent infection model with spatial heterogeneity
- Mathematical modeling and optimal control for COVID‐19 with population behavior
- An epidemiological model for analysing pandemic trends of novel coronavirus transmission with optimal control
- Stability analysis of fuzzy HTLV-I infection model: a dynamic approach
- Bifurcation analysis and optimal control of COVID-19 with exogenous reinfection and media coverages
- Stability analysis of general delayed HTLV-I dynamics model with mitosis and CTL immunity
- Dynamics of an HTLV-I infection model with delayed CTLs immune response
- Stationary distribution and extinction of a stochastic HLIV model with viral production and Ornstein-Uhlenbeck process
- Global dynamics analysis for a nonlinear HTLV-I model with logistic proliferation and CTL response
- Modelling lockdown measures in epidemic outbreaks using selective socio-economic containment with uncertainty
- Stability analysis and numerical simulations of the fractional COVID-19 pandemic model
- Global dynamics analysis of non-local delayed reaction-diffusion Avian influenza model with vaccination and multiple transmission routes in the spatial heterogeneous environment
- Global stability analysis of viral infection model with logistic growth rate, general incidence function and cellular immunity
- Threshold dynamics of an HIV-1 model with both virus-to-cell and cell-to-cell transmissions, immune responses, and three delays
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