To cure or not to cure: consequences of immunological interactions in CML treatment
DOI10.1101/494575zbMATH Open1417.92069OpenAlexW2905498810WikidataQ91681433 ScholiaQ91681433MaRDI QIDQ2002115FDOQ2002115
Authors: Artur César Fassoni, Ingo Roeder, Ingmar Glauche 
Publication date: 11 July 2019
Published in: Bulletin of Mathematical Biology (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1101/494575
Recommendations
- Stability analysis of a model of interaction between the immune system and cancer cells in chronic myelogenous leukemia
- A mathematical model for chronic myelogenous leukemia (CML) and T cell interaction
- A mathematical model of Imatinib and interferon-Alpha combined treatment of chronic myeloid leukemia
- Stability analysis of a mathematical model for chronic myeloid leukemia eradication
- A quantitative study on the role of TKI combined with Wnt/\(\beta\)-catenin signaling and IFN-\(\alpha\) in the treatment of CML through deterministic and stochastic approaches
bifurcationsmathematical modelingordinary differential equationschronic myeloid leukemiaimmunological controltreatment-free remission
Medical applications (general) (92C50) Qualitative investigation and simulation of ordinary differential equation models (34C60)
Cites Work
- Nonlinear dynamics of immunogenic tumors: Parameter estimation and global bifurcation analysis
- Interactions between the immune system and cancer: A brief review of non-spatial mathematical models
- Algorithms in real algebraic geometry
- A mathematical model for chronic myelogenous leukemia (CML) and T cell interaction
- Strategic treatment interruptions during imatinib treatment of chronic myelogenous leukemia
- Post-transplantation dynamics of the immune response to chronic myelogenous leukemia
- Stability analysis of a model of interaction between the immune system and cancer cells in chronic myelogenous leukemia
Cited In (8)
- Strategic treatment interruptions during imatinib treatment of chronic myelogenous leukemia
- Dynamics and bifurcations in a model of chronic myeloid leukemia with optimal immune response windows
- Tumor microenvironment as a metapopulation model: the effects of angiogenesis, emigration and treatment modalities
- Modelling of killer T-cell and cancer cell subpopulation dynamics under immuno- and chemotherapies
- Questions from the fourth son: a clinician reflects on immunomonitoring, surrogate markers and systems biology
- Post-transplantation dynamics of the immune response to chronic myelogenous leukemia
- Ultimate dynamics analysis of the 5D structural leukemia model and partitioning of the parameter space
- Influence of the age structure on the stability in a tumor-immune model for chronic myeloid leukemia
This page was built for publication: To cure or not to cure: consequences of immunological interactions in CML treatment
Report a bug (only for logged in users!)Click here to report a bug for this page (MaRDI item Q2002115)
