To cure or not to cure: consequences of immunological interactions in CML treatment

DOI10.1101/494575MaRDI QIDQ2002115zbMATH OpenOpenAlexWikidataFDO

Authors Artur César Fassoni, Ingo Roeder, Ingmar Glauche

Publication date 11 July 2019

Published in Bulletin of Mathematical Biology (Search for Journal in Brave)

Copyright license Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International

Full work available at URL https://doi.org/10.1101/494575

zbMATH Keywords

bifurcationsmathematical modelingordinary differential equationschronic myeloid leukemiaimmunological controltreatment-free remission

Mathematics Subject Classification ID

Medical applications (general) (92C50) Qualitative investigation and simulation of ordinary differential equation models (34C60)

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Cites work

• A mathematical model for chronic myelogenous leukemia (CML) and T cell interaction
• Algorithms in real algebraic geometry
• Interactions between the immune system and cancer: A brief review of non-spatial mathematical models
• Nonlinear dynamics of immunogenic tumors: Parameter estimation and global bifurcation analysis
• Post-transplantation dynamics of the immune response to chronic myelogenous leukemia
• Stability analysis of a model of interaction between the immune system and cancer cells in chronic myelogenous leukemia
• Strategic treatment interruptions during imatinib treatment of chronic myelogenous leukemia

Cited in

(8)

• Dynamics and bifurcations in a model of chronic myeloid leukemia with optimal immune response windows
• Modelling of killer T-cell and cancer cell subpopulation dynamics under immuno- and chemotherapies
• Post-transplantation dynamics of the immune response to chronic myelogenous leukemia
• Strategic treatment interruptions during imatinib treatment of chronic myelogenous leukemia
• Ultimate dynamics analysis of the 5D structural leukemia model and partitioning of the parameter space
• Questions from the fourth son: a clinician reflects on immunomonitoring, surrogate markers and systems biology
• Tumor microenvironment as a metapopulation model: the effects of angiogenesis, emigration and treatment modalities
• Influence of the age structure on the stability in a tumor-immune model for chronic myeloid leukemia

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