Halting the hallmarks: a cellular automaton model of early cancer growth inhibition

DOI10.1007/S11047-015-9508-3MaRDI QIDQ2003442zbMATH OpenOpenAlexFDO

Authors Jenna Butler, Frances Mackay, Colin Denniston, Mark Daley

Publication date 8 July 2019

Published in Natural Computing (Search for Journal in Brave)

Full work available at URL https://ir.lib.uwo.ca/brainpub/150

zbMATH Keywords

cellular automatacancer modellinglattice Boltzmannbinary fluidcancer hallmarks

Mathematics Subject Classification ID

Medical applications (general) (92C50) Cellular automata (computational aspects) (68Q80)

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Cites work

• A Model for Collision Processes in Gases. I. Small Amplitude Processes in Charged and Neutral One-Component Systems
• A hybrid cellular automaton model of clonal evolution in cancer: the emergence of the glycolytic phenotype
• An evolutionary hybrid cellular automaton model of solid tumour growth
• An ordinary differential equation model for the multistep transformation to cancer
• Computational analysis of the influence of the microenvironment on carcinogenesis
• Development of a mathematical model that predicts the outcome of hormone therapy for prostate cancer
• Evolution of cell motility in an individual-based model of tumour growth
• LATTICE BOLTZMANN METHOD FOR FLUID FLOWS
• Mathematical modelling of cancer cell invasion of tissue
• Modelling of cancer growth, evolution and invasion: bridging scales and models
• Patient-calibrated agent-based modelling of ductal carcinoma in situ (DCIS): from microscopic measurements to macroscopic predictions of clinical progression
• Selected topics in cancer modeling. Genesis, evolution, immune competition, and therapy
• The Use of Hybrid Cellular Automaton Models for Improving Cancer Therapy
• The lattice Boltzmann equation. For fluid dynamics and beyond

Cited in

(1)

• Avoiding healthy cells extinction in a cancer model

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