Modeling the stem cell hypothesis: investigating the effects of cancer stem cells and TGF - on tumor growth
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Modeling the stem cell hypothesis: investigating the effects of cancer stem cells and TGF \(- \beta\) on tumor growth
Modeling the stem cell hypothesis: investigating the effects of cancer stem cells and TGF \(- \beta\) on tumor growth
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Cites work
- A mathematical model of the enhancement of tumor vaccine efficacy by immunotherapy
- A mathematical model of tumor-immune evasion and siRNA treatment
- A methodology for performing global uncertainty and sensitivity analysis in systems biology
- A multicompartment mathematical model of cancer stem cell-driven tumor growth dynamics
- Mathematical biology: modeling and analysis
- Mathematical modelling of plasticity and phenotype switching in cancer cell populations
Cited in
(16)- A stem cell model for carcinogenesis
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- Investigating the link between epithelial-mesenchymal transition and the cancer stem cell phenotype: a mathematical approach
- Strategies for cancer stem cell elimination: insights from mathematical modeling
- A computational model for investigating tumor apoptosis induced by mesenchymal stem cell-derived secretome
- On tumoural growth and treatment under cellular dedifferentiation
- Mathematical modeling of tumor and cancer stem cells treated with CAR-T therapy and inhibition of TGF-\(\beta\)
- Mathematical modelling of cancer stem cell-targeted immunotherapy
- Cancer stem cell, niche and EGFR decide tumor development and treatment response: a bio-computational simulation study
- Modeling the treatment of glioblastoma multiforme and cancer stem cells with ordinary differential equations
- Mathematical modeling of pancreatic cancer treatment with cancer stem cells
- Stability analysis of mathematical models for nonlinear growth kinetics of breast cancer stem cells
- Mathematical characterization of heterogeneity in a cancer stem cell driven tumor growth model with nonlinear self-renewal
- Mathematical approach to predict the drug effects on cancer stem cell models
- Effects of a differentiating therapy on cancer-stem-cell-driven tumors
- Mathematical modelling of plasticity and phenotype switching in cancer cell populations
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