Bifurcations in a dynamical model of the innate immune system response to initial pulmonary infection
DOI10.1007/S12346-022-00573-2zbMATH Open1492.34062OpenAlexW4214600942MaRDI QIDQ2114407FDOQ2114407
Authors: Juan Su, Min Lu, Jicai Huang
Publication date: 15 March 2022
Published in: Qualitative Theory of Dynamical Systems (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1007/s12346-022-00573-2
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degenerate Hopf bifurcationresultant eliminationdegenerate Bogdanov-Takens bifurcationinnate immunepulmonary infection
Medical applications (general) (92C50) Qualitative investigation and simulation of ordinary differential equation models (34C60) Topological structure of integral curves, singular points, limit cycles of ordinary differential equations (34C05) Bifurcation theory for ordinary differential equations (34C23) Homoclinic and heteroclinic solutions to ordinary differential equations (34C37) Stability of solutions to ordinary differential equations (34D20)
Cites Work
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- Codimension 3 B-T bifurcations in an epidemic model with a nonlinear incidence
- Generic 3-parameter families of vector fields on the plane, unfolding a singularity with nilpotent linear part. The cusp case of codimension 3
- Bifurcation analysis of a predator-prey system with generalised Holling type III functional response
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- Decomposition of algebraic sets and applications to weak centers of cubic systems
- A low dimensional dynamical model of the initial pulmonary innate response to infection
- Bifurcation analysis of a dynamical model for the innate immune response to initial pulmonary infections
Cited In (8)
- A low dimensional dynamical model of the initial pulmonary innate response to infection
- High codimension bifurcations analysis in a predator-prey system with Michaelis-Menten type predator harvesting
- Bifurcation analysis of a dynamical model for the innate immune response to initial pulmonary infections
- Title not available (Why is that?)
- Backward bifurcation in neutrophil-pathogen interaction
- Mathematical model of a three-stage innate immune response to a pneumococcal lung infection
- Analysis of Lauffenburger-Kennedy bacterial infection model for tissue inflammation dynamics
- Modeling the bistable dynamics of the innate immune system
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