Metabolic implications for the mechanism of mitochondrial endosymbiosis and human hereditary disorders

DOI10.1016/J.JTBI.2007.04.017MaRDI QIDQ2211547zbMATH OpenOpenAlexWikidataFDO

Authors Benjamin Lovegren de Bivort, Chun-Chung Chen, Fabrizio Perretti, G. Negro, Thomas M. Philip, Y. Bar-Yam

Publication date 11 November 2020

Published in Journal of Theoretical Biology (Search for Journal in Brave)

Full work available at URL https://doi.org/10.1016/j.jtbi.2007.04.017

zbMATH Keywords

evolutionmutualismmitochondriamultilevel selectionendosymbiosis

Mathematics Subject Classification ID

Cell biology (92C37) Pathology, pathophysiology (92C32)

Recommendations

• An information approach to mitochondrial dysfunction. Extending Swerdlow's hypothesis
• A thermodynamic approach to energy transduction in mitochondria
• A comparison of mtDNA deletion mutant proliferation mechanisms
• Modeling the evolution of the human mitochondrial genome
• Genetics of metabolic resistance
• Metabolic involutions

Cites work

• scientific article; zbMATH DE number 3835792 (Why is no real title available?)
• scientific article; zbMATH DE number 896049 (Why is no real title available?)
• scientific article; zbMATH DE number 3106184 (Why is no real title available?)
• Equilibrium points in n -person games
• Game theory

This page was built for publication: Metabolic implications for the mechanism of mitochondrial endosymbiosis and human hereditary disorders

Report a bug (only for logged in users!)Click here to report a bug for this page (MaRDI item Q2211547)