Neutrophil dynamics in response to chemotherapy and G-CSF
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Publication:2263503
DOI10.1016/J.JTBI.2011.10.017zbMATH Open1307.92147OpenAlexW2050948710WikidataQ50603649 ScholiaQ50603649MaRDI QIDQ2263503FDOQ2263503
Authors: Changjing Zhuge, Jinzhi Lei, M. C. Mackey
Publication date: 18 March 2015
Published in: Journal of Theoretical Biology (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1016/j.jtbi.2011.10.017
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Cites Work
- Understanding cyclical thrombocytopenia: a mathematical modeling approach
- Oscillations in cyclical neutropenia: new evidence based on mathematical modeling
- A mathematical model of hematopoiesis. I: Periodic chronic myelogenous leukemia
- A mathematical model of hematopoiesis. II: Cyclical neutropenia
- Multistability in an age-structured model of hematopoiesis: cyclical neutropenia
- Modelling human granulopoiesis under poly-chemotherapy with G-CSF support
- Mathematical model for G-CSF administration after chemotherapy
- The complex effect of granulocyte colony-stimulating factor on human granulopoiesis analyzed by a new physiologically-based mathematical model
- G-CSF control of neutrophils dynamics in the blood
- Title not available (Why is that?)
Cited In (14)
- Bistability and state transition of a delay differential equation model of neutrophil dynamics
- Mathematical model for G-CSF administration after chemotherapy
- Neutrophil dynamics after chemotherapy and G-CSF: the role of pharmacokinetics in shaping the response
- Blood cell dynamics: half of a century of modelling
- Response of an oscillatory differential delay equation to a periodic stimulus
- The complex effect of granulocyte colony-stimulating factor on human granulopoiesis analyzed by a new physiologically-based mathematical model
- G-CSF control of neutrophils dynamics in the blood
- Understanding, treating and avoiding hematological disease: better medicine through mathematics?
- Global dynamics of delay recruitment models with maximized lifespan
- A mathematical model of chromosome recombination-induced drug resistance in cancer therapy
- Modelling human granulopoiesis under poly-chemotherapy with G-CSF support
- Characterizing chemotherapy-induced neutropenia and monocytopenia through mathematical modelling
- Neutrophil dynamics during concurrent chemotherapy and G-CSF administration: mathematical modelling guides dose optimisation to minimise neutropenia
- Origins of oscillation patterns in cyclical thrombocytopenia
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