An allelopathy based model for the \textit{Listeria} overgrowth phenomenon

DOI10.1016/J.MBS.2013.10.008MaRDI QIDQ2441601zbMATH OpenOpenAlexWikidataFDO

Authors Hedia Fgaier, Martin Kalmokoff, Timothy Ells, Hermann J. Eberl

Publication date 25 March 2014

Published in Mathematical Biosciences (Search for Journal in Brave)

Full work available at URL https://doi.org/10.1016/j.mbs.2013.10.008

zbMATH Keywords

inhibitionListeria innocuaListeria monocytogenes

Mathematics Subject Classification ID

Cell biology (92C37) Physiological, cellular and medical topics (92C99) Ecology (92D40) Application models in control theory (93C95)

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Cites work

• A chemostat model of resource competition and allelopathy
• scientific article; zbMATH DE number 417962 (Why is no real title available?)
• Ordinary differential equations. An introduction
• Parameter identification and quantitative comparison of differential equations that describe physiological adaptation of a bacterial population under iron limitation
• The Theory of the Chemostat

Cited in

(13)

• Mathematical model of interaction between bacteriocin-producing lactic acid bacteria and listeria. Part II: bifurcations and applications
• With-in host dynamics of \textit{L. monocytogenes} and thresholds for distinct infection scenarios
• A theoretical model of listeriosis driven by cross contamination of ready-to-eat food products
• The operating diagram of a model of two competitors in a chemostat with an external inhibitor
• Global analysis of a model of competition in the chemostat with internal inhibitor
• A competition model in the chemostat with allelopathy and substrate inhibition
• Thermodynamic inhibition in chemostat models. With an application to bioreduction of uranium
• Mathematical model of interaction between bacteriocin-producing lactic acid bacteria and listeria. I: Steady states and thresholds
• A density-dependent model of competition for one resource in the chemostat
• Substrate inhibition can produce coexistence and limit cycles in the chemostat model with allelopathy
• Mortality can induce limit cycles in a chemostat competition model between plasmid-bearing and plasmid-free organisms with allelopathy
• A competition model in the chemostat between plasmid-bearing and plasmid-free organisms with an internal inhibitor and different removal rates
• A mixed-culture biofilm model with cross-diffusion

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