Global properties for a virus dynamics model with lytic and non-lytic immune responses, and nonlinear immune attack rates
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Publication:2933562
DOI10.1142/S021833901450017XzbMATH Open1302.92057OpenAlexW2050401523MaRDI QIDQ2933562FDOQ2933562
Authors: Cruz Vargas-De-León 
Publication date: 3 December 2014
Published in: Journal of Biological Systems (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1142/s021833901450017x
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Cited In (16)
- Modeling the dynamics of viral-host interaction during treatment of productively infected cells and free virus involving total immune response
- GLOBAL PROPERTIES OF A MODEL OF IMMUNE EFFECTOR RESPONSES TO VIRAL INFECTIONS
- Complete dynamical analysis for a nonlinear HTLV-I infection model with distributed delay, CTL response and immune impairment
- Mathematical analysis of a cell mediated immunity in a virus dynamics model with nonlinear infection rate and removal
- Stochastic virus infection model with Ornstein-Uhlenbeck perturbation: extinction and stationary distribution analysis
- Global properties of nonlinear humoral immunity viral infection models
- Stability of a mathematical model of tumour-induced angiogenesis
- Stability and Hopf bifurcation of a CTL-inclusive HIV-1 infection model with both viral and cellular infections, and three delays
- Global stability of infectious disease models with contact rate as a function of prevalence index
- Stability of an adaptive immunity viral infection model with multi-stages of infected cells and two routes of infection
- Global stability in a viral infection model with lytic and nonlytic immune responses
- Qualitative investigation of cytolytic and noncytolytic immune response in an HBV model
- Complete global dynamics of a delayed viral infection model with lytic and nonlytic effectors
- Viral dynamics of delayed CTL-inclusive HIV-1 infection model with both virus-to-cell and cell-to-cell transmissions
- LYAPUNOV FUNCTION AND GLOBAL PROPERTIES OF VIRUS DYNAMICS WITH CTL IMMUNE RESPONSE
- Global properties of a virus dynamics model with self-proliferation of CTLs
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