Scaling theory for anomalous semiclassical quantum transport
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Publication:2994589
DOI10.1088/1751-8113/49/4/045101zbMATH Open1348.82076arXiv1512.06948OpenAlexW2216373058MaRDI QIDQ2994589FDOQ2994589
Authors: M. I. Sena-Junior, A. M. S. Macêdo 
Publication date: 2 August 2016
Published in: Journal of Physics A: Mathematical and Theoretical (Search for Journal in Brave)
Abstract: Quantum transport through devices coupled to electron reservoirs can be described in terms of the full counting statistics (FCS) of charge transfer. Transport observables, such as conductance and shot-noise power are just cumulants of FCS and can be obtained from the sample's average density of transmission eigenvalues, which in turn can be obtained from a finite element representation of the saddle-point equation of the Keldysh (or supersymmetric) non-linear sigma-model, known as quantum circuit theory. Normal universal metallic behavior in the semiclassical regime is controlled by the presence of a Fabry-Perot singularity in the average density of transmission eigenvalues. We present general conditions for the suppression of Fabry-Perot modes in the semiclassical regime in a sample of arbitrary shape, a disordered conductor or a network of ballistic quantum dots, which leads to an anomalous metallic phase. Through a double-scaling limit, we derive a scaling equation for anomalous metallic transport, in the form of a nonlinear differential equation, which generalizes the ballistic-diffusive scaling equation of a normal metal. The two-parameter stationary solution of our scaling equation generalizes Dorokhov's universal single-parameter distribution of transmission eigenvalues. We provide a simple interpretation of the stationary solution using a thermodynamic analogy with a spin-glass system. As an application, we consider a system formed by a diffusive wire coupled via a barrier to normal-superconductor (NS) reservoirs. We observe anomalous reflectionless tunneling, when all perfectly transmitting channels are suppressed, which cannot be explained by the usual mechanism of disorder-induced opening of tunneling channels.
Full work available at URL: https://arxiv.org/abs/1512.06948
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