Analysis of Lauffenburger-Kennedy bacterial infection model for tissue inflammation dynamics

DOI10.1080/17513758.2018.1538463zbMATH Open1447.92497OpenAlexW2899149818WikidataQ58076304 ScholiaQ58076304MaRDI QIDQ3300969FDOQ3300969

Authors: Yu Yang, Alhaji Cherif, Yu-Xin Zhang 

Publication date: 31 July 2020

Published in: Journal of Biological Dynamics (Search for Journal in Brave)

Full work available at URL: https://doi.org/10.1080/17513758.2018.1538463

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zbMATH Keywords

numerical resultsHopf bifurcationssteady statesLauffenburger-Kennedy bacterial infection model

Mathematics Subject Classification ID

Epidemiology (92D30) Bifurcation theory for ordinary differential equations (34C23)

Cites Work

• Transient behavior of a chemotaxis system modelling certain types of tissue inflammation
• Analysis of a lumped model for tissue inflammation dynamics
• Qualitative analysis of a mathematical model for tissue inflammation dynamics
• Title not available (Why is that?)
• The resolution of inflammation: a mathematical model of neutrophil and macrophage interactions
• Overview of mathematical approaches used to model bacterial chemotaxis. II: Bacterial popu\-lations
• Diffusion-driven instability and bifurcation in the Lengyel-Epstein system
• Development and applications of a model for cellular response to multiple chemotactic cues
• PARAMETER DOMAINS FOR GENERATING SPATIAL PATTERN: A COMPARISON OF REACTION–DIFFUSION AND CELL-CHEMOTAXIS MODELS
• Localized bacterial infection in a distributed model for tissue inflammation
• Analysis of chemotactic bacterial distributions in population migration assays using a mathematical model applicable to steep or shallow attractant gradients
• Modeling endotoxin-induced systemic inflammation using an indirect response approach
• Analysis of the effects of immune cell motility and chemotaxis on target elimination dynamics
• Title not available (Why is that?)

Cited In (3)

• Mathematical analysis of a model describing the invasion of bacteria in burn wounds
• Localized bacterial infection in a distributed model for tissue inflammation
• The dynamics of bacterial infection, innate immune response, and antibiotic treatment

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