Global stability for a discrete epidemic model for disease with immunity and latency spreading in a heterogeneous host population
DOI10.1016/J.NONRWA.2011.07.031zbMATH Open1238.34108OpenAlexW2109448849MaRDI QIDQ420040FDOQ420040
Authors: Yoshiaki Muroya, Alfredo Bellen, Yoichi Enatsu, Yukihiko Nakata
Publication date: 20 May 2012
Published in: Nonlinear Analysis. Real World Applications (Search for Journal in Brave)
Full work available at URL: http://hdl.handle.net/20.500.11824/417
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Cited In (25)
- Global stability of a multi-group SIS epidemic model with varying total population size
- Asymptotically non-expansive self-maps and global stability with ultimate boundedness of dynamic systems
- Bifurcation analysis of a discrete \({SIRS}\) epidemic model with standard incidence rate
- A non-standard finite difference scheme for a delayed and diffusive viral infection model with general nonlinear incidence rate
- Global dynamics in a class of discrete-time epidemic models with disease courses
- Almost periodic solutions of a discrete mutualism model with variable delays
- Global threshold property in an epidemic model for disease with latency spreading in a heterogeneous host population
- The existence of codimension-two bifurcation in a discrete SIS epidemic model with standard incidence
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- Stability and bifurcation analysis of a discrete severe fever with thrombocytopenia syndrome model
- Global stability of a multi-group SIS epidemic model for population migration
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- Global dynamics for a class of discrete SEIRS epidemic models with general nonlinear incidence
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- A nonstandard finite difference method applied to a mathematical cholera model
- A diffusive SVEIR epidemic model with time delay and general incidence
- Bifurcation analysis and chaos control in a discrete epidemic system
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- Stability and codimension 2 bifurcations of a discrete time SIR model
- Global stability of a delayed and diffusive virus model with nonlinear infection function
- Nonstandard finite difference scheme for a diffusive within-host virus dynamics model with both virus-to-cell and cell-to-cell transmissions
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