Global properties of a delayed HIV infection model with CTL immune response
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Cites work
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Cited in
(46)- A class of delayed viral models with saturation infection rate and immune response
- Global stability of HIV-1 infection model with two time delays
- Stability and Hopf bifurcation for a five-dimensional virus infection model with Beddington-DeAngelis incidence and three delays
- Stability of pathogen dynamics models with viral and cellular infections and immune impairment
- Hopf bifurcation analysis of nonlinear HIV infection model and the effect of delayed immune response with drug therapies
- Analyzing global stability of a viral model with general incidence rate and cytotoxic T lymphocytes immune response
- Global stability of HIV infection of CD4\(^{+}\) T cells and macrophages with CTL immune response and distributed delays
- Global stability of delayed viral infection models with nonlinear antibody and CTL immune responses and general incidence rate
- Modeling within-host viral dynamics: the role of CTL immune responses in the evolution of drug resistance
- Optimal control of an HIV model with CTL cells and latently infected cells
- Mathematical analysis of an HIV latent infection model including both virus-to-cell infection and cell-to-cell transmission
- Dynamics of an HIV model with multiple infection stages and treatment with different drug classes
- GLOBAL PROPERTIES OF HIV DYNAMICS MODELS INCLUDING IMPAIRMENT OF B-CELL FUNCTIONS
- Modeling the effect of reactive oxygen species and CTL immune response on HIV dynamics
- Global properties of a cell mediated immunity in HIV infection model with two classes of target cells and distributed delays
- Stability analysis of a virus infection model with humoral immunity response and two time delays
- Global properties for a class of latent HIV infection dynamics model with CTL immune response
- Analysis of HIV models with multiple target cell populations and general nonlinear rates of viral infection and cell death
- Stability and Hopf bifurcation of HIV-1 model with Holling II infection rate and immune delay
- Global stability of a delayed HIV-1 infection model with absorption and CTL immune response
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- A global stability analysis for a delayed HIV infection model with humoral immune response
- A within-host virus model with multiple infected stages under time-varying environments
- Dynamics analysis of a delayed HIV infection model with CTL immune response and antibody immune response
- Global behavior of delay differential equations model of HIV infection with apoptosis
- Dynamics of cytotoxic T-lymphocytes and helper cells in human immunodeficiency virus infection with Hill-type infection rate and sigmoidal CTL expansion
- Stability of delayed HIV dynamics models with two latent reservoirs and immune impairment
- Stability and feedback stabilization of HIV infection model with two classes of target cells
- Stability analysis of HIV-1 model with multiple delays
- Stability of virus infection models with antibodies and chronically infected cells
- Optimal multi-therapeutic HIV treatment using a global optimal switching scheme
- Global stability of a diffusive and delayed virus dynamics model with Crowley-Martin incidence function and CTL immune response
- Stability analysis of a model for HBV infection with cure of infected cells and intracellular delay
- Global dynamics of a delayed HIV-1 infection model with CTL immune response
- Effect of cellular reservoirs and delays on the global dynamics of HIV
- Stability and Hopf bifurcation analysis for an HIV infection model with Beddington-DeAngelis incidence and two delays
- Threshold dynamics of HIV-1 virus model with cell-to-cell transmission, cell-mediated immune responses and distributed delay
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- Stability and Hopf bifurcation of an HIV infection model with saturation incidence and two delays
- Dynamics of an HIV infection model with two time delays
- Modeling and stability analysis of HIV/HTLV-I co-infection
- Global stability of a diffusive and delayed virus infection model with general incidence function and adaptive immune response
- Age-structured within-host HIV dynamics with multiple target cells
- Global stability of a general virus dynamics model with multi-staged infected progression and humoral immunity
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