The stability and Hopf bifurcation for an HIV model with saturated infection rate and double delays
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Publication:4636792
DOI10.1142/S1793524518500407zbMATH Open1385.92051WikidataQ130105429 ScholiaQ130105429MaRDI QIDQ4636792FDOQ4636792
Authors: Ying Lv, Zhixing Hu, Fucheng Liao
Publication date: 20 April 2018
Published in: International Journal of Biomathematics (Search for Journal in Brave)
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Medical epidemiology (92C60) Epidemiology (92D30) Stability theory of functional-differential equations (34K20) Bifurcation theory of functional-differential equations (34K18)
Cites Work
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- Global stability and Hopf bifurcation of an HIV-1 infection model with saturation incidence and delayed CTL immune response
- A differential equation model of HIV infection of CD4\(^+\) \(T\)-cells with cure rate
- Global stability of an HIV pathogenesis model with cure rate
- Global stability for an SEIR epidemiological model with varying infectivity and infinite delay
- The trade-off between mutual interference and time lags in predator-prey systems
- On the zeros of a fourth degree exponential polynomial with applications to a neural network model with delays
- Stability and Hopf bifurcation in a viral infection model with nonlinear incidence rate and delayed immune response
- Stability and Hopf bifurcation in a delayed model for HIV infection of CD\(4^+\) T cells
- Analysis of an HIV infection model with treatments and delayed immune response
Cited In (8)
- Dynamics of an HIV infection model with two time delays
- Dynamical analysis of antigen-driven t-cell infection model with multiple delays
- Hopf bifurcation analysis of nonlinear HIV infection model and the effect of delayed immune response with drug therapies
- Stability analysis of an HIV pathogenesis model with saturating infection rate and time delay
- Dynamics of two delays differential equations model of HIV pathogenesis with absorption and saturation incidence
- Bifurcation and optimal control analysis of delayed models for Huanglongbing
- Accounting for multi-delay effects in an HIV-1 infection model with saturated infection rate, recovery and proliferation of host cells
- Stability and Hopf bifurcation of an HIV infection model with saturation incidence and two delays
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