A multicompartment mathematical model of cancer stem cell-driven tumor growth dynamics
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Publication:467723
DOI10.1007/S11538-014-9976-0zbMATH Open1300.92045OpenAlexW2057776488WikidataQ34076143 ScholiaQ34076143MaRDI QIDQ467723FDOQ467723
Authors: Suzanne L. Weekes, Brian Barker, Sarah Bober, Karina Cisneros, Justina Cline, Amanda Thompson, Philip Hahnfeldt, Heiko Enderling, Lynn Hlatky 
Publication date: 5 November 2014
Published in: Bulletin of Mathematical Biology (Search for Journal in Brave)
Full work available at URL: http://europepmc.org/articles/pmc4140966
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Cites Work
- On the proportion of cancer stem cells in a tumour
- Mathematical modelling of cancer stem cells population behavior
- The tumor growth paradox and immune system-mediated selection for cancer stem cells
- Multispecies model of cell lineages and feedback control in solid tumors
- The universal properties of stem cells as pinpointed by a simple discrete model
- Cancer stem cells as the engine of unstable tumor progression
- A mathematical model of cancer stem cell lineage population dynamics with mutation accumulation and telomere length hierarchies
Cited In (37)
- On tumoural growth and treatment under cellular dedifferentiation
- Mathematical modeling of bone marrow -- peripheral blood dynamics in the disease state based on current emerging paradigms. I
- A mathematical model of cancer stem cell lineage population dynamics with mutation accumulation and telomere length hierarchies
- Dynamics and optimal control of an SEIAQR epidemic model with media coverage
- Some Results on the Population Behavior of Cancer Stem Cells
- The tumor growth paradox and immune system-mediated selection for cancer stem cells
- Well-posedness of a coupled PDE-ODE model of stem cell lineage involving homeostatic regulation
- Using a stem cell and progeny model to illustrate the relationship between cell cycle times of in vivo human tumour cell tissue populations, in vitro primary cultures and the cell lines derived from them
- Mathematical modeling of tumor and cancer stem cells treated with CAR-T therapy and inhibition of TGF-\(\beta\)
- A non-local model for cancer stem cells and the tumour growth paradox
- On the proportion of cancer stem cells in a tumour
- The overshoot and phenotypic equilibrium in characterizing cancer dynamics of reversible phenotypic plasticity
- Modeling the cancer stem cell hypothesis
- Mathematical analysis on a diffusion model describing the compatibility between two types of tumor cells
- Control of cell fraction and population recovery during tissue regeneration in stem cell lineages
- Mathematical modelling of plasticity and phenotype switching in cancer cell populations
- Mass concentration in a nonlocal model of clonal selection
- Replicator dynamics of cancer stem cell: selection in the presence of differentiation and plasticity
- Modeling approaches to studying stem cells in cancer
- Characterizing inhibited tumor growth in stem-cell-driven non-spatial cancers
- Mathematical modeling of pancreatic cancer treatment with cancer stem cells
- Mathematical modelling of cancer stem cell-targeted immunotherapy
- Modelling of cancer stem cell driven solid tumors
- Multispecies model of cell lineages and feedback control in solid tumors
- Growth of tumours with stem cells: the effect of crowding and ageing of cells
- Modeling the stem cell hypothesis: investigating the effects of cancer stem cells and TGF \(- \beta\) on tumor growth
- A mathematical study of the impact of cell plasticity on tumour control probability
- Mathematical characterization of heterogeneity in a cancer stem cell driven tumor growth model with nonlinear self-renewal
- Effect of feedback regulation on stem cell fractions in tissues and tumors: understanding chemoresistance in cancer
- Cellular feedback networks and their resilience against mutations
- Negligible long-term impact of nonlinear growth dynamics on heterogeneity in models of cancer cell populations
- Self-sustaining sub-populations of progenitor cells
- Cancer stem cells as the engine of unstable tumor progression
- A stochastic population model for the impact of cancer cell dormancy on therapy success
- Modeling tumorspheres reveals cancer stem cell niche building and plasticity
- On a mathematical model of tumor growth based on cancer stem cells
- Mathematical modeling of bone marrow -- peripheral blood dynamics in the disease state based on current emerging paradigms. II
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