Modelling and finite-time stability analysis of psoriasis pathogenesis
From MaRDI portal
Publication:5348358
Recommendations
- A control-based mathematical study on psoriasis dynamics with special emphasis on \(\text{IL}-21\) and \(\text{IFN} - \gamma\) interaction network
- Saturation effects on immunopathogenic mechanism of psoriasis: a theoretical approach
- Mathematical models for therapeutic approaches to control psoriasis
- A vivid cytokines interaction model on psoriasis with the effect of impulse biologic (\(\mathrm{TNF}-\alpha\) inhibitor) therapy
- Mathematical insights on psoriasis regulation: role of \(\mathrm{Th}_{1}\) and \(\mathrm{Th}_{2}\) cells
Cites work
Cited in
(9)- A vivid cytokines interaction model on psoriasis with the effect of impulse biologic (\(\mathrm{TNF}-\alpha\) inhibitor) therapy
- Modelling of indirect cell-cell interaction networks mediated by IFN\(\gamma\)/IL-4 cytokine involved in atopic dermatitis
- A model analysis to measure the adherence of Etanercept and Fezakinumab therapy for the treatment of psoriasis
- Fractional-order model of the disease psoriasis: a control based mathematical approach
- Mathematical insights on psoriasis regulation: role of \(\mathrm{Th}_{1}\) and \(\mathrm{Th}_{2}\) cells
- Optimal control problems for a mathematical model of the treatment of psoriasis
- A model of the optimal immunotherapy of psoriasis by introducing IL-10 and IL-22 inhibitors
- On a third-order singular arc of optimal control in a minimization problem for a mathematical model of psoriasis treatment
- Saturation effects on immunopathogenic mechanism of psoriasis: a theoretical approach
This page was built for publication: Modelling and finite-time stability analysis of psoriasis pathogenesis
Report a bug (only for logged in users!)Click here to report a bug for this page (MaRDI item Q5348358)