Uniform boundedness and eventual Hölder continuity to a cancer invasion model with remodeling of ECM and nonlinear diffusion
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ECM remodelingHölder continuitylong time behaviorMoser-type iterationnonlinear diffusionuniform boundedness
Asymptotic behavior of solutions to PDEs (35B40) A priori estimates in context of PDEs (35B45) Smoothness and regularity of solutions to PDEs (35B65) Initial-boundary value problems for second-order parabolic systems (35K51) Quasilinear parabolic equations (35K59) PDEs in connection with biology, chemistry and other natural sciences (35Q92)
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Cites work
- A chemotaxis-haptotaxis model: the roles of nonlinear diffusion and logistic source
- A user's guide to PDE models for chemotaxis
- Asymptotic stability in a quasilinear chemotaxis-haptotaxis model with general logistic source and nonlinear signal production
- Boundedness of solutions to a quasilinear higher-dimensional chemotaxis-haptotaxis model with nonlinear diffusion
- Elliptic and parabolic equations
- Energy-type estimates and global solvability in a two-dimensional chemotaxis-haptotaxis model with remodeling of non-diffusible attractant
- From 1970 until present: The Keller-Segel model in chemotaxis and its consequences. I
- Global bounded weak solutions and asymptotic behavior to a chemotaxis-Stokes model with non-Newtonian filtration slow diffusion
- Global boundedness of solutions to a chemotaxis-haptotaxis model with tissue remodeling
- Global classical solution and boundedness to a chemotaxis-haptotaxis model with re-establishment mechanisms
- Global existence of a two-dimensional chemotaxis-haptotaxis model with remodeling of non-diffusible attractant
- Global solvability and stabilization to a cancer invasion model with remodelling of ECM
- Global weak solutions in a PDE-ODE system modeling multiscale cancer cell invasion
- scientific article; zbMATH DE number 1061253 (Why is no real title available?)
- Initiation of slime mold aggregation viewed as an instability
- MATHEMATICAL MODELLING OF CANCER CELL INVASION OF TISSUE: THE ROLE OF THE UROKINASE PLASMINOGEN ACTIVATION SYSTEM
- Mathematical modelling of cancer invasion of tissue: dynamic heterogeneity
- Model for chemotaxis
- Negligibility of haptotaxis effect in a chemotaxis-haptotaxis model
- Nondegeneracy of blow-up points for the parabolic Keller-Segel system
- Stabilization to a cancer invasion model with remodeling mechanism and slow diffusion
- Toward a mathematical theory of Keller-Segel models of pattern formation in biological tissues
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