Global dynamics of a multiscale model for hepatitis C virus infection

DOI10.1016/J.AML.2023.108904MaRDI QIDQ6141005zbMATH OpenFDO

Authors Xia Wang, Xin Meng, Libin Rong

Publication date 2 January 2024

Published in Applied Mathematics Letters (Search for Journal in Brave)

Full work available at URL https://doi.org/10.1016/J.AML.2023.108904

zbMATH Keywords

cell-to-cell transmissionglobal stabilityHCVimmune responsesmultiscale model

Mathematics Subject Classification ID

Qualitative investigation and simulation of ordinary differential equation models (34C60) PDEs in connection with biology, chemistry and other natural sciences (35Q92) Epidemiology (92D30)

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Cites work

• A PDE multiscale model of hepatitis C virus infection can be transformed to a system of ODEs
• HIV low viral load persistence under treatment: insights from a model of cell-to-cell viral transmission
• Mathematical analysis of a transformed ODE from a PDE multiscale model of hepatitis C virus infection
• Mathematical analysis of an HIV latent infection model including both virus-to-cell infection and cell-to-cell transmission
• Mathematical analysis of multiscale models for hepatitis C virus dynamics under therapy with direct-acting antiviral agents
• Mathematical models of immune effector responses to viral infections: Virus control versus the development of pathology
• Threshold dynamics of an HIV infection model with two distinct cell subsets
• Threshold dynamics of HCV model with cell-to-cell transmission and a non-cytolytic cure in the presence of humoral immunity

Cited in

(4)

• Stochastic two-strain epidemic model with saturated incidence rates driven by Lévy noise
• Dynamics of an age-structured multiscale hepatitis C virus model with two infection modes and antibody immune response
• Rich dynamics of a hepatitis C virus infection model with logistic proliferation and time delays
• Global stability analysis of a nested multiscale model for intracellular and intercellular dynamics

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