Periodic insulin stimulation of Akt: dynamic steady states and robustness
From MaRDI portal
Publication:6154147
DOI10.1016/j.mbs.2023.109113OpenAlexW4389312147MaRDI QIDQ6154147
Adelle C. F. Coster, Catheryn W. Gray
Publication date: 14 February 2024
Published in: Mathematical Biosciences (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1016/j.mbs.2023.109113
Physiology (general) (92C30) Qualitative investigation and simulation of ordinary differential equation models (34C60)
Cites Work
- A calcium-based phantom bursting model for pancreatic islets
- From insulin to Akt: time delays and dominant processes
- A molecular mathematical model of glucose mobilization and uptake
- Transitions between bursting modes in the integrated oscillator model for pancreatic \(\beta\)-cells
- Crosstalk in transition: the translocation of Akt
- A model for ultradian oscillations of insulin and glucose
- Modeling the glucose-insulin regulatory system and ultradian insulin secretory oscillations with two explicit time delays
- Mathematical investigation of diabetically impaired ultradian oscillations in the glucose-insulin regulation
- Deconstructing the integrated oscillator model for pancreatic \(\beta \)-cells
This page was built for publication: Periodic insulin stimulation of Akt: dynamic steady states and robustness
