A mathematical model for HIV dynamics with multiple infections: implications for immune escape
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Topological structure of integral curves, singular points, limit cycles of ordinary differential equations (34C05) Qualitative investigation and simulation of ordinary differential equation models (34C60) Asymptotic properties of solutions to ordinary differential equations (34D05) Stability of solutions to ordinary differential equations (34D20) Global stability of solutions to ordinary differential equations (34D23) Medical epidemiology (92C60) Epidemiology (92D30)
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Cites work
- A two-sex model of human papillomavirus infection: vaccination strategies and a case study
- An epidemic model in a patchy environment
- Analysis of an HIV model with immune responses and cell-to-cell transmission
- Bistability analysis of an HIV model with immune response
- Conflict and accord of optimal treatment strategies for HIV infection within and between hosts
- Convergence results and a Poincaré-Bendixson trichotomy for asymptotically autonomous differential equations
- Differential equation models of some parasitic infections: Methods for the study of asymptotic behavior
- Dynamical behavior of a vector-host epidemic model with demographic structure
- Dynamics of HIV infection of CD4^ + T cells
- Dynamics of HIV-1 coinfection in different susceptible target cell populations during cell-free infection
- Emergence of HIV-1 drug resistance during antiretroviral treatment
- HIV low viral load persistence under treatment: insights from a model of cell-to-cell viral transmission
- Mathematical modeling of escape of HIV from cytotoxic t lymphocyte responses
- Modeling HIV dynamics under combination therapy with inducers and antibodies
- Modeling HIV multiple infection
- Modeling multiple infection of cells by viruses: challenges and insights
- Modeling the role of acquired immune response and antiretroviral therapy in the dynamics of HIV infection
- Modeling the role of macrophages in HIV persistence during antiretroviral therapy
- Modeling within-host viral dynamics: the role of CTL immune responses in the evolution of drug resistance
- Persistence under Relaxed Point-Dissipativity (with Application to an Endemic Model)
- Reproduction numbers and sub-threshold endemic equilibria for compartmental models of disease transmission
- Threshold dynamics of HIV-1 virus model with cell-to-cell transmission, cell-mediated immune responses and distributed delay
- Towards a new combination therapy with vectored immunoprophylaxis for HIV: modeling ``Shock and kill strategy
- Virus dynamics in the presence of synaptic transmission
Cited in
(3)- Bistability and bifurcation analysis in a viral infection model with fully coupled logistic cell growth
- Bifurcation and stability analysis of within host HIV dynamics with multiple infections and intracellular delay
- Exploring the potential for persistent HIV infection through dynamical analysis of models incorporating homeostatic proliferation of CD8+ T cells
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