On drug therapy for an HIV infection age model with cellular and immune delays

DOI10.58997/EJDE.CONF.27.T1MaRDI QIDQ6599378zbMATH OpenFDO

Authors Nicoleta Tarfulea

Publication date 6 September 2024

Published in Electronic Journal of Differential Equations (EJDE) (Search for Journal in Brave)

zbMATH Keywords

HIV infectioninhibitorsdrug therapyviral age model

Mathematics Subject Classification ID

Medical applications (general) (92C50) Epidemiology (92D30) Growth, boundedness, comparison of solutions to functional-differential equations (34K12)

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Cites work

• scientific article; zbMATH DE number 7004012 (Why is no real title available?)
• A CTL-INCLUSIVE MATHEMATICAL MODEL FOR ANTIRETROVIRAL TREATMENT OF HIV INFECTION
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• A mathematical model for CTL effect on drug resistence during antiretroviral treatment of HIV infection
• A mathematical model for HIV treatment with time-varying antiretroviral therapy
• A mathematical model for the emergence of HIV drug resistance during periodic bang-bang type antiretroviral treatment
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• A model of HIV-1 infection with two time delays: mathematical analysis and comparison with patient data
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• Estimating kinetic parameters from HIV primary infection data through the eyes of three different mathematical models
• Global dynamics of an in-host viral model with intracellular delay
• Global stability of an HIV-1 model with distributed intracellular delays and a combination therapy
• HIV-1 infection and low steady state viral loads
• Mathematical Analysis of HIV-1 Dynamics in Vivo
• Modeling HIV-1 virus dynamics with both virus-to-cell infection and cell-to-cell transmission
• Modeling pharmacodynamics on HIV latent infection: choice of drugs is key to successful cure via early therapy
• Periodic oscillations in leukopoiesis models with two delays
• Stability analysis in delayed within-host viral dynamics with both viral and cellular infections

Cited in

(2)

• The effects of delay and impulsive drug therapy in an HIV model with CTLs immune response
• Optimal drug therapy in a multi-pathways HIV-1 infection model with immune response delay

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