Dynamical analysis of a delayed reaction-diffusion virus infection model with logistic growth and humoral immune impairment
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Publication:721776
DOI10.1016/J.CHAOS.2018.03.006zbMATH Open1391.35385OpenAlexW2796881328WikidataQ130040175 ScholiaQ130040175MaRDI QIDQ721776FDOQ721776
Authors: Hui Miao, Xamxinur Abdurahman, Long Zhang, Zhidong Teng 
Publication date: 20 July 2018
Published in: Chaos, Solitons and Fractals (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1016/j.chaos.2018.03.006
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Cited In (16)
- A class of virus dynamic model with inhibitory effect on the growth of uninfected T cells caused by infected T cells and its stability analysis
- Spatiotemporal dynamics of a delayed viral infection model with two modes of transmission and immune impairment
- Global properties of virus dynamics with B-cell impairment
- Analysis of a diffusive virus infection model with humoral immunity, cell-to-cell transmission and nonlinear incidence
- Impact of B-cell impairment on virus dynamics with time delay and two modes of transmission
- A reaction-diffusion model for nested within-host and between-host dynamics in an environmentally-driven infectious disease
- Dynamics analysis of a delayed viral infection model with immune impairment
- Dynamics of a nonlocal dispersal in‐host viral model with humoral immunity
- Lyapunov functional for virus infection model with diffusion and state-dependent delays
- GLOBAL PROPERTIES OF HIV DYNAMICS MODELS INCLUDING IMPAIRMENT OF B-CELL FUNCTIONS
- NSFD scheme and dynamic consistency of a delayed diffusive humoral immunity viral infection model
- Global dynamics of a delayed latent virus model with both virus-to-cell and cell-to-cell transmissions and humoral immunity
- Stability of a general HIV-1 reaction-diffusion model with multiple delays and immune response
- Spatial dynamics of a viral infection model with immune response and nonlinear incidence
- Analysis and simulation of a delayed HIV model with reaction-diffusion and sliding control
- Threshold dynamics of an HIV-1 model with both virus-to-cell and cell-to-cell transmissions, immune responses, and three delays
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