Contact inhibition of growth described using a multiphase model and an individual cell based model
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Publication:735102
DOI10.1016/J.AML.2008.06.051zbMATH Open1171.92314OpenAlexW2017726699MaRDI QIDQ735102FDOQ735102
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Cites work
- A HYBRID MODEL FOR TUMOR SPHEROID GROWTH IN VITRO I: THEORETICAL DEVELOPMENT AND EARLY RESULTS
- A hybrid mathematical model of solid tumour invasion: the importance of cell adhesion
- COARSE GRAINING IN SIMULATED CELL POPULATIONS
- How cellular movement determines the collective force generated by the \textit{Dictyostelium discoideum} slug
- Mathematical modelling of the loss of tissue compression responsiveness and its role in solid tumour development
- Multiphase modelling of tumour growth and extracellular matrix interaction: mathematical tools and applications
- ON THE CLOSURE OF MASS BALANCE MODELS FOR TUMOR GROWTH
Cited in
(13)- The influence of anisotropic growth and geometry on the stress of solid tumors
- Evaluating the influence of mechanical stress on anticancer treatments through a multiphase porous media model
- Are tumor cell lineages solely shaped by mechanical forces?
- Patient-calibrated agent-based modelling of ductal carcinoma in situ (DCIS): from microscopic measurements to macroscopic predictions of clinical progression
- AN IN VITRO CELL POPULATION DYNAMICS MODEL INCORPORATING CELL SIZE, QUIESCENCE, AND CONTACT INHIBITION
- Quantifying the relationship between cell division angle and morphogenesis through computational modeling
- Modeling contact inhibition of growth: traveling waves
- On the concentration profile of a growth inhibitory factor in multicell spheroids
- Minimal cellular automaton model with heterogeneous cell sizes predicts epithelial colony growth
- A computational model of cell membrane bioelectric polarization and depolarization, connected with cell proliferation, in different tissue geometries
- Negative Control of Cell Proliferation. Growth Arrest versus Apoptosis. Role of βGBP
- Model of vascular desmoplastic multispecies tumor growth
- The effects of cell compressibility, motility and contact inhibition on the growth of tumor cell clusters using the cellular Potts model
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