The arrested immunity hypothesis in an immunoepidemiological model of Chlamydia transmission
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Publication:743530
DOI10.1016/J.TPB.2014.01.005zbMATH Open1296.92129OpenAlexW1970961755WikidataQ37844964 ScholiaQ37844964MaRDI QIDQ743530FDOQ743530
David M. Vickers, Nathaniel D. Osgood
Publication date: 24 September 2014
Published in: Theoretical Population Biology (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1016/j.tpb.2014.01.005
Cites Work
- Title not available (Why is that?)
- A mathematical model for the investigation of the Th1 immune response to \textit{Chlamydia trachomatis}
- Evaluating the importance of within- and between-host selection pressures on the evolution of chronic pathogens
- Modeling regulation mechanisms in the immune system
- Human sexual contact network as a bipartite graph
- Persistence of viral infections on the population level explained by an immunoepidemiological model
- An in-host model of acute infection: measles as a case study
- A mathematical model of chlamydial infection incorporating movement of chlamydial particles
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