Mathematical model of the impact of a nonantibiotic treatment for Clostridium difficile on the endemic prevalence of vancomycin-resistant enterococci in a hospital setting
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Publication:764186
DOI10.1155/2012/605861zbMATH Open1234.92029OpenAlexW1968579034WikidataQ35691700 ScholiaQ35691700MaRDI QIDQ764186FDOQ764186
Authors: Daniel T. Grima, E. M. C. D'Agata, Glenn Webb 
Publication date: 13 March 2012
Published in: Computational \& Mathematical Methods in Medicine (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1155/2012/605861
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Cited In (9)
- Stationary solution of a stochastic nosocomial epidemic model in hospital intensive care units
- Mathematical modelling reveals properties of tcdc required for it to be a negative regulator of toxin production in \textit{clostridium difficile}
- Vancomycin-resistant enterococci colonization-infection model: parameter impacts and outbreak risks
- Mathematical modeling of the effects of nutrient competition and bile acid metabolism by the gut microbiota on colonization resistance against \textit{Clostridium difficile}
- Mathematically modeling the effect of touch frequency on the environmental transmission of \textit{Clostridioides difficile} in healthcare settings
- No appendix necessary: fecal transplants and antibiotics can resolve \textit{Clostridium difficile} infection
- Optimal control of vancomycin-resistant enterococci using preventive care and treatment of infections
- The work of Glenn F. Webb
- Individual based models and differential equations models of nosocomial epidemics in hospital intensive care units
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