Model of coupled transient changes of Rac, Rho, adhesions and stress fibers alignment in endothelial cells responding to shear stress

DOI10.1016/J.JTBI.2004.09.004zbMATH Open1442.92044OpenAlexW2023699847WikidataQ42471564 ScholiaQ42471564MaRDI QIDQ776556FDOQ776556

Authors: D. Kharzeev 

Publication date: 9 July 2020

Published in: Journal of Theoretical Biology (Search for Journal in Brave)

Full work available at URL: https://doi.org/10.1016/j.jtbi.2004.09.004

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zbMATH Keywords

endothelial cellsmathematical modelstress fibersfocal adhesionRho GTPases

Mathematics Subject Classification ID

Biomechanics (92C10) Cell biology (92C37)

Cites Work

• Selecting a common direction. II: Peak-like solutions representing total alignment of cell clusters
• Title not available (Why is that?)
• An Integrodifferential Model for Orientational Distributions of F-actin in Cells
• Stress-induced alignment of actin filaments and the mechanics of cytogel
• Model for the alignment of actin filaments in endothelial cells subjected to fluid shear stress

Cited In (6)

• Switching behaviour in vascular smooth muscle cell-matrix adhesion during oscillatory loading
• Passive and active fiber reorientation in anisotropic materials
• A mathematical model of the coupled mechanisms of cell adhesion, contraction and spreading
• Cell orientation under stretch: stability of a linear viscoelastic model
• A model of localised Rac1 activation in endothelial cells due to fluid flow
• A kinematic model coupling stress fiber dynamics with JNK activation in response to matrix stretching

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