Partial equilibrium approximations in apoptosis. II: The death-inducing signaling complex subsystem

DOI10.1016/J.MBS.2015.10.009MaRDI QIDQ899346zbMATH OpenOpenAlexWikidataFDO

Authors Ya-Jing Huang, Liu Hong, Wen-An Yong

Publication date 28 December 2015

Published in Mathematical Biosciences (Search for Journal in Brave)

Full work available at URL https://doi.org/10.1016/j.mbs.2015.10.009

zbMATH Keywords

apoptosisbiochemical reactionspartial equilibrium approximationprinciple of detailed balance

Mathematics Subject Classification ID

Cell biology (92C37) Biochemistry, molecular biology (92C40)

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Cites work

• A mathematical model of receptor-mediated apoptosis: Dying to know why FASL is a trimer
• An interesting class of partial differential equations
• Analysis of Kinetic Reaction Mechanisms
• Computational cell biology
• scientific article; zbMATH DE number 1796810 (Why is no real title available?)
• Mathematical physiology
• Partial equilibrium approximations in apoptosis. I: The intracellular-signaling subsystem
• Quasi steady state and partial equilibrium approximations: their relation and their validity
• Recasting nonlinear differential equations as S-systems: a canonical nonlinear form
• Simple computational models of type I/type II cells in Fas signaling-induced apoptosis
• Singular perturbations of first-order hyperbolic systems with stiff source terms

Cited in

(9)

• Terminality implies no-signalling\dots and much more than that
• Simple computational models of type I/type II cells in Fas signaling-induced apoptosis
• Partial equilibrium approximations in apoptosis. I: The intracellular-signaling subsystem
• Equilibria and stability of a class of positive feedback loops. Mathematical analysis and its application to caspase-dependent apoptosis
• Model reduction of the intracellular-signaling subsystem of apoptosis
• DISCRETE NONDETERMINISTIC MODELING OF THE FAS PATHWAY
• scientific article; zbMATH DE number 5501422 (Why is no real title available?)
• Markov Modelling of Mitochondrial BAK Activation Kinetics during Apoptosis
• EQUILIBRIUM, STABILITY AND DYNAMICAL RESPONSE IN A MODEL OF THE EXTRINSIC APOPTOSIS PATHWAY

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