Partial equilibrium approximations in apoptosis. II: The death-inducing signaling complex subsystem
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Publication:899346
DOI10.1016/J.MBS.2015.10.009zbMATH Open1364.92008OpenAlexW2216349887WikidataQ46645374 ScholiaQ46645374MaRDI QIDQ899346FDOQ899346
Wen-An Yong, Liu Hong, Ya-Jing Huang
Publication date: 28 December 2015
Published in: Mathematical Biosciences (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1016/j.mbs.2015.10.009
Cites Work
- Mathematical physiology
- Simple computational models of type I/type II cells in Fas signaling-induced apoptosis
- Partial equilibrium approximations in apoptosis. I: The intracellular-signaling subsystem
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- Singular perturbations of first-order hyperbolic systems with stiff source terms
- An interesting class of partial differential equations
- Computational cell biology
- Analysis of Kinetic Reaction Mechanisms
- Quasi steady state and partial equilibrium approximations: their relation and their validity
- Recasting nonlinear differential equations as S-systems: a canonical nonlinear form
- A mathematical model of receptor-mediated apoptosis: Dying to know why FASL is a trimer
Cited In (5)
- Title not available (Why is that?)
- Markov Modelling of Mitochondrial BAK Activation Kinetics during Apoptosis
- EQUILIBRIUM, STABILITY AND DYNAMICAL RESPONSE IN A MODEL OF THE EXTRINSIC APOPTOSIS PATHWAY
- Terminality implies no-signalling\dots and much more than that
- Equilibria and stability of a class of positive feedback loops. Mathematical analysis and its application to caspase-dependent apoptosis
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