Modeling genetic architecture: A multilinear theory of gene interaction
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Publication:1597399
DOI10.1006/TPBI.2000.1508zbMATH Open0994.92029OpenAlexW2011145067WikidataQ73598608 ScholiaQ73598608MaRDI QIDQ1597399FDOQ1597399
Authors: Thomas F. Hansen, Günter P. Wagner 
Publication date: 10 October 2002
Published in: Theoretical Population Biology (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1006/tpbi.2000.1508
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Cites Work
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- Evolution of gene networks by gene duplications: a mathematical model and its implications on genome organization.
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- Equilibria in an epistatic viability model under arbitrary strength of selection
Cited In (24)
- Genetical ESS-models. I. Concepts and basic model
- Evolutionary dynamics in the two-locus two-allele model with weak selection
- The E(NK) model: Extending the NK model to incorporate gene-by-environment interactions and epistasis for diploid genomes
- A new model of estimating genotypic values in quantitative inheritance
- Mathematical frameworks for phenotypical selection and epistasis
- Estimating genetic architectures from artificial-selection responses: a random-effect framework
- Epistasis can increase multivariate trait diversity in haploid non-recombining populations
- On the Fourier transform of a quantitative trait: implications for compressive sensing
- Variance component analysis of polymorphic metabolic systems
- Multilocus population-genetic theory
- On the evolution of epistasis. II: A generalized Wright-Kimura framework
- HAMILTONIAN MODEL FOR MULTIDIMENSIONAL EPISTASIS
- On the evolution of epistasis. III: The haploid case with mutation
- Effects of epistasis and the evolution of genetic architecture: exact results for a 2-locus model
- Selective mating in a continuous model of epistasis
- Analysis of phenotype-genotype associations using genomic informational field theory (GIFT)
- A simple model of co-evolutionary dynamics caused by epistatic selection
- The mapping of epistatic effects onto a genealogical tree in haploid populations
- The role of epistatic gene interactions in the response to selection and the evolution of evolvabil\-i\-ty
- Evolvability and robustness: a paradox restored
- A computational method to detect epistatic effects contributing to a quantitative trait
- A simple fitness proxy for structured populations with continuous traits, with case studies on the evolution of haplo-diploids and genetic dimorphisms
- A mathematical framework for evo-devo dynamics
- On the evolution of epistasis. I: Diploids under selection
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