Modeling genetic architecture: A multilinear theory of gene interaction
From MaRDI portal
(Redirected from Publication:1597399)
Recommendations
- Mathematical frameworks for phenotypical selection and epistasis
- Effects of epistasis and the evolution of genetic architecture: exact results for a 2-locus model
- The role of epistatic gene interactions in the response to selection and the evolution of evolvabil\-i\-ty
- Quantitative variability and multilocus polymorphism under epistatic selection
- Epistasis in the multiple locus symmetric viability model
Cites work
- A stochastic model concerning the maintenance of genetic variability in quantitative characters.
- Deleterious mutations, variable epistatic interactions, and the evolution of recombination
- Epistasis and pleiotropy as natural properties of transcriptional regulation
- Equilibria in an epistatic viability model under arbitrary strength of selection
- Evolution of gene networks by gene duplications: a mathematical model and its implications on genome organization.
- scientific article; zbMATH DE number 53687 (Why is no real title available?)
- scientific article; zbMATH DE number 3493681 (Why is no real title available?)
- scientific article; zbMATH DE number 3218370 (Why is no real title available?)
- Landscapes and their correlation functions
- Quantitative variability and multilocus polymorphism under epistatic selection
Cited in
(26)- Genetical ESS-models. I. Concepts and basic model
- Estimating genetic architectures from artificial-selection responses: a random-effect framework
- Evolutionary dynamics in the two-locus two-allele model with weak selection
- Evolvability and robustness: a paradox restored
- A simple model of co-evolutionary dynamics caused by epistatic selection
- The E(NK) model: Extending the NK model to incorporate gene-by-environment interactions and epistasis for diploid genomes
- On the Fourier transform of a quantitative trait: implications for compressive sensing
- Analysis of phenotype-genotype associations using genomic informational field theory (GIFT)
- Mathematical frameworks for phenotypical selection and epistasis
- Multilocus population-genetic theory
- A computational method to detect epistatic effects contributing to a quantitative trait
- Variance component analysis of polymorphic metabolic systems
- The mapping of epistatic effects onto a genealogical tree in haploid populations
- The role of epistatic gene interactions in the response to selection and the evolution of evolvabil\-i\-ty
- On the evolution of epistasis. I: Diploids under selection
- Epistasis can increase multivariate trait diversity in haploid non-recombining populations
- scientific article; zbMATH DE number 1104068 (Why is no real title available?)
- Can nonlinear epigenetic interactions obscure causal relations between genotype and phenotype?
- A simple fitness proxy for structured populations with continuous traits, with case studies on the evolution of haplo-diploids and genetic dimorphisms
- A new model of estimating genotypic values in quantitative inheritance
- HAMILTONIAN MODEL FOR MULTIDIMENSIONAL EPISTASIS
- On the evolution of epistasis. III: The haploid case with mutation
- Effects of epistasis and the evolution of genetic architecture: exact results for a 2-locus model
- A mathematical framework for evo-devo dynamics
- Selective mating in a continuous model of epistasis
- On the evolution of epistasis. II: A generalized Wright-Kimura framework
This page was built for publication: Modeling genetic architecture: A multilinear theory of gene interaction
Report a bug (only for logged in users!)Click here to report a bug for this page (MaRDI item Q1597399)
