Mathematical models of retinitis pigmentosa: the oxygen toxicity hypothesis
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Cites work
- A mathematical model for photoreceptor interactions
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- Mathematical model of the role of RdCVF in the coexistence of rods and cones in a healthy eye
- One-hit stochastic decline in a mechanochemical model of cytoskeleton-induced neuron death. I: Cell-fate arrival times
- One-hit stochastic decline in a mechanochemical model of cytoskeleton-induced neuron death. II: Transition state metastability
- One-hit stochastic decline in a mechanochemical model of cytoskeleton-induced neuron death. III: Diffusion pulse death zones
- Optimal control in the treatment of retinitis pigmentosa
- Photoreceptor death: spatiotemporal patterns arising from one-hit death kinetics and a diffusible cell death factor
- Quantifying the metabolic contribution to photoreceptor death in retinitis pigmentosa via a mathematical model
- Scale-free neurodegeneration: cellular heterogeneity and the stretched exponential kinetics of cell death
- Tracing the progression of retinitis pigmentosa via photoreceptor interactions
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(11)- Tracing the progression of retinitis pigmentosa via photoreceptor interactions
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- Blood flow regulation and oxygen transport in a heterogeneous model of the mouse retina
- Photoreceptor death: spatiotemporal patterns arising from one-hit death kinetics and a diffusible cell death factor
- Optimal control in the treatment of retinitis pigmentosa
- MODELLING HYDROGEN CLEARANCE FROM THE RETINA
- scientific article; zbMATH DE number 7478914 (Why is no real title available?)
- Optimal control with MANF treatment of photoreceptor degeneration
- Spatially-dependent model for rods and cones in the retina
- Quantifying the metabolic contribution to photoreceptor death in retinitis pigmentosa via a mathematical model
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