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Cites work
- scientific article; zbMATH DE number 2038872 (Why is no real title available?)
- scientific article; zbMATH DE number 2150813 (Why is no real title available?)
- A survey of models for tumor-immune system dynamics
- Kolmogorov and population dynamics
- Mathematical modeling of cancer radiovirotherapy
- Modeling of cancer virotherapy with recombinant measles viruses
- Population models with singular equilibrium
Cited in
(44)- A mathematical model for cell cycle-specific cancer virotherapy
- New virus dynamics in the presence of multiple infection
- Modeling dynamics of cancer radiovirotherapy
- Modelling the spatiotemporal dynamics of chemovirotherapy cancer treatment
- Backward Hopf bifurcation in a mathematical model for oncolytic virotherapy with the infection delay and innate immune effects
- Dynamics of an oncolytic virus model
- Hopf bifurcation and normal form in a delayed oncolytic model
- Enhancing oncolytic virotherapy: observations from a Voronoi cell-based model
- Dynamical behavior of a new oncolytic virotherapy model based on gene variation
- Interlocked feedback loops balance the adaptive immune response
- A mathematical model verifying potent oncolytic efficacy of M1 virus
- Use slow-spread of oncolytic virus to depress exponential growth of tumor cells
- Slow-fast model and therapy optimization for oncolytic treatment of tumors
- Computational modeling of cancer response to oncolytic virotherapy: improving the effectiveness of viral spread and anti tumor efficacy
- Memory versus effector immune responses in oncolytic virotherapies
- The role of viral dynamics and infectivity in models of oncolytic virotherapy for tumours with different motility
- Basic stochastic model for tumor virotherapy
- Hopf bifurcation in oncolytic therapeutic modeling: viruses as anti-tumor means with viral lytic cycle
- Quantitative impact of immunomodulation versus oncolysis with cytokine-expressing virus therapeutics
- A mathematical model for oncolytic virus spread using the telegraph equation
- A mathematical model of oncolytic virotherapy with time delay
- Bifurcation analysis of the cancer virotherapy system with time delay and diffusion
- Finding Hopf bifurcation islands and identifying thresholds for success or failure in oncolytic viral therapy
- A phenotype-structured mathematical model for the influence of hypoxia on oncolytic virotherapy
- Treatment strategies for combining immunostimulatory oncolytic virus therapeutics with dendritic cell injections
- Mathematical modeling of viral infection dynamics in spherical organs
- On a mathematical model of tumor-immune system interactions with an oncolytic virus therapy
- Hopf bifurcation without parameters in deterministic and stochastic modeling of cancer virotherapy. I
- Developing a minimally structured mathematical model of cancer treatment with oncolytic viruses and dendritic cell injections
- The role of the innate immune system in oncolytic virotherapy
- Computational modeling approaches to studying the dynamics of oncolytic viruses
- A general non-local delay model on oncolytic virus therapy
- The role of viral infectivity in oncolytic virotherapy outcomes: a mathematical study
- A hybrid discrete-continuum modelling approach for the interactions of the immune system with oncolytic viral infections
- Oncolytic virotherapy for tumours following a Gompertz growth law
- Modeling oncolytic virus therapy with distributed delay and nonlocal diffusion
- Modeling oncolytic virotherapy: is complete tumor-tropism too much of a good thing?
- Control exponential growth of tumor cells with slow spread of oncolytic virus
- Car T-cell and oncolytic virus dynamics and determinants of combination therapy success for glioblastoma
- Oncolytic virus treatment of human breast cancer cells: modelling therapy efficacy
- Hopf bifurcation analysis of a tumor virotherapy model with two time delays
- Agent-based and continuum models for spatial dynamics of infection by oncolytic viruses
- Turing bifurcation induced by cross-diffusion and amplitude equation in oncolytic therapeutic model: viruses as anti-tumor means
- Modelling heterogeneity in viral-tumour dynamics: the effects of gene-attenuation on viral characteristics
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