Survival analysis for tumor cells in stochastic switching environment

DOI10.1016/J.AMC.2019.04.010MaRDI QIDQ2009586zbMATH OpenOpenAlexWikidataFDO

Authors Dongxi Li, Yue Zhao

Publication date 29 November 2019

Published in Applied Mathematics and Computation (Search for Journal in Brave)

Full work available at URL https://doi.org/10.1016/j.amc.2019.04.010

zbMATH Keywords

extinctiontelegraph noisetumorstochastic switching environment

Mathematics Subject Classification ID

Systems biology, networks (92C42) Stochastic ordinary differential equations (aspects of stochastic analysis) (60H10) Cell biology (92C37)

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Cites work

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• scientific article; zbMATH DE number 2062257 (Why is no real title available?)
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• Distributed delays in a hybrid model of tumor-immune system interplay
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• Long time behavior of a tumor-immune system competition model perturbed by environmental noise
• Noise-induced phenomena in the environmental sciences
• Nonlinear dynamics of immunogenic tumors: Parameter estimation and global bifurcation analysis
• On the relation between white shot noise, Gaussian white noise, and the dichotomic Markov process
• Persistence and extinction in stochastic non-autonomous logistic systems
• Phase diagrams of noise induced transitions -- exact results for a class of external coloured noise.
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Cited in

(11)

• Most probable trajectories in a two-dimensional tumor-immune system under stochastic perturbation
• Dynamic behavior of stochastic prostate cancer system with comprehensive therapy under regime switching
• Hazard function for cancer patients and cancer cell dynamics
• Long time behavior of a tumor-immune system competition model perturbed by environmental noise
• The interplay between stochastic gene switching and certain metronomics therapies.
• Statistical inference of the rates of cell proliferation and phenotypic switching in cancer
• The threshold of stochastic tumor-immune model with regime switching
• Survival analysis for tumor growth model with stochastic perturbation
• Dynamical behaviors of a tumor-immune-vitamin model with random perturbation
• Tumor stabilization induced by t-cell recruitment fluctuations
• Optimal control strategy for cancer remission using combinatorial therapy: a mathematical model-based approach

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