A Turing mechanism in order to explain the patchy nature of Crohn's disease
From MaRDI portal
Publication:2040279
DOI10.1007/s00285-021-01635-wzbMath1466.92004arXiv2007.13587OpenAlexW3178270859WikidataQ113905492 ScholiaQ113905492MaRDI QIDQ2040279
Eric Ogier-Denis, Ana I. Toledo, Hatem Zaag, Gregoire Nadin
Publication date: 13 July 2021
Published in: Journal of Mathematical Biology (Search for Journal in Brave)
Full work available at URL: https://arxiv.org/abs/2007.13587
PDEs in connection with biology, chemistry and other natural sciences (35Q92) Medical applications (general) (92C50) Mathematical modeling or simulation for problems pertaining to biology (92-10)
Related Items (1)
Cites Work
- A PDE approach of inflammatory phase dynamics in diabetic wounds
- Reaction-diffusion model of atherosclerosis development
- The resolution of inflammation: a mathematical model of neutrophil and macrophage interactions
- Localized bacterial infection in a distributed model for tissue inflammation
- Initiation of slime mold aggregation viewed as an instability
- Bifurcation and dynamics in a mathematical model of early atherosclerosis. How acute inflammation drives lesion development
- General model of inflammation
- Analysis of a lumped model for tissue inflammation dynamics
- Model of colonic inflammation: immune modulatory mechanisms in inflammatory bowel disease
- The dynamics of acute inflammation
- A reduced mathematical model of the acute inflammatory response. I: Derivation of model and analysis of anti-inflammation
- A reduced mathematical model of the acute inflammatory response. II: Capturing scenarios of repeated endotoxin administration
- Parabolic equations in biology. Growth, reaction, movement and diffusion
- Atherosclerosis Initiation Modeled as an Inflammatory Process
- Random walk with persistence and external bias
- Pattern Formation in a Model of Acute Inflammation
- A mathematical model of atherogenesis as an inflammatory response
- The chemical basis of morphogenesis
This page was built for publication: A Turing mechanism in order to explain the patchy nature of Crohn's disease
