A reduced mathematical model of the acute inflammatory response. II: Capturing scenarios of repeated endotoxin administration

DOI10.1016/J.JTBI.2006.02.015MaRDI QIDQ2201848zbMATH OpenOpenAlexWikidataFDO

Authors Judy Day, Jonathan Rubin, Yoram Vodovotz, Carson C. Chow, Angela Reynolds, Gilles Clermont

Publication date 17 September 2020

Published in Journal of Theoretical Biology (Search for Journal in Brave)

Full work available at URL https://doi.org/10.1016/j.jtbi.2006.02.015

zbMATH Keywords

immunologymathematical modelinginflammationanti-inflammatory cytokinesendotoxin tolerance

Mathematics Subject Classification ID

Pathology, pathophysiology (92C32)

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Cites work

• Simulating, Analyzing, and Animating Dynamical Systems
• The dynamics of acute inflammation
• Two-dimensional global manifolds of vector fields

Cited in

(26)

• A reduced mathematical model of the acute inflammatory response. I: Derivation of model and analysis of anti-inflammation
• Mathematical model of triple-negative breast cancer in response to combination chemotherapies
• A model of TLR4 signaling and tolerance using a qualitative, particle-event-based method: Introduction of spatially configured stochastic reaction chambers (SCSRC)
• Integrated inflammatory stress (ITIS) model
• Immune therapeutic strategies using optimal controls with \(L^{1}\) and \(L^{2}\) type objectives
• Modeling the effects of systemic mediators on the inflammatory phase of wound healing
• A Turing mechanism in order to explain the patchy nature of Crohn's disease
• A boundary value approach to optimization with an application to salmonella competition
• A unified computational model for the human response to lipopolysaccharide-induced inflammation
• Global sensitivity analysis of a mathematical model of acute inflammation identifies nonlinear dependence of cumulative tissue damage on host interleukin-6 responses
• A simple mathematical model of signaling resulting from the binding of lipopolysaccharide with toll-like receptor 4 demonstrates inherent preconditioning behavior
• A mathematical model of the inflammatory response to pathogen challenge
• A dual negative regulation model of Toll-like receptor 4 signaling for endotoxin preconditioning in human endotoxemia
• Modeling endotoxin-induced systemic inflammation using an indirect response approach
• Mathematical modeling of energy consumption in the acute inflammatory response
• On the Nonexistence of Terrestrial Canards: Linking Canards and Rivers
• A quantitative model of thermal injury-induced acute inflammation
• Modeling the bistable dynamics of the innate immune system
• An ensemble of models of the acute inflammatory response to bacterial lipopolysaccharide in rats: results from parameter space reduction
• A mathematical model of pulmonary gas exchange under inflammatory stress
• Global sensitivity analysis used to interpret biological experimental results
• Personalized mathematical model of endotoxin-induced inflammatory responses in young men and associated changes in heart rate variability
• A mathematical model of the use of supplemental oxygen to combat surgical site infection
• A systems-theoretical framework for health and disease: inflammation and preconditioning from an abstract modeling point of view
• Toward a model-free feedback control synthesis for treating acute inflammation
• Mechanistic simulations of inflammation: current state and future prospects

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• XPPAUT

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