A unified computational model for the human response to lipopolysaccharide-induced inflammation
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Publication:6609967
DOI10.1007/16618_2022_39MaRDI QIDQ6609967FDOQ6609967
Authors: Kristen A. Windoloski, Elisabeth O. Bangsgaard, Atanaska Dobreva, Johnny T. Ottesen, Mette S. Olufsen 
Publication date: 24 September 2024
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ordinary differential equationsdosing strategiesinflammationtime scalescomputational modelsystems modelendotoxin responselipopolysaccharide (LPS)
Cites Work
- A simple mathematical model of signaling resulting from the binding of lipopolysaccharide with toll-like receptor 4 demonstrates inherent preconditioning behavior
- The dynamics of acute inflammation
- A reduced mathematical model of the acute inflammatory response. I: Derivation of model and analysis of anti-inflammation
- Mathematical modeling of the hypothalamic-pituitary-adrenal gland (HPA) axis, including hippocampal mechanisms
- Patient-specific modeling of the neuroendocrine HPA-axis and its relation to depression: ultradian and circadian oscillations
- Modeling endotoxin-induced systemic inflammation using an indirect response approach
- A reduced mathematical model of the acute inflammatory response. II: Capturing scenarios of repeated endotoxin administration
- Modeling the bistable dynamics of the innate immune system
- Modeling the influence of circadian rhythms on the acute inflammatory response
- Modeling the hypothalamus-pituitary-adrenal axis: A review and extension
- Integrated inflammatory stress (ITIS) model
- Patient specific modeling of the HPA axis related to clinical diagnosis of depression
- Cardiovascular dynamics during head-up tilt assessed via pulsatile and non-pulsatile models
- Personalized mathematical model of endotoxin-induced inflammatory responses in young men and associated changes in heart rate variability
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