Global dynamics of an age-structured within-host viral infection model with cell-to-cell transmission and general humoral immunity response
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Publication:2045787
DOI10.3934/MBE.2020075zbMATH Open1470.92095OpenAlexW2991415878WikidataQ90792506 ScholiaQ90792506MaRDI QIDQ2045787FDOQ2045787
Authors: Ran Zhang, Shengqiang Liu 
Publication date: 13 August 2021
Published in: Mathematical Biosciences and Engineering (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.3934/mbe.2020075
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Cited In (16)
- Global analysis of age-structured within-host virus model
- Global dynamics of an age-structured in-host viral infection model with humoral immunity
- Global properties of an age-structured virus model with saturated antibody-immune response, multi-target cells, and general incidence rate
- Hopf Bifurcation of an Age-Structured Epidemic Model with Quarantine and Temporary Immunity Effects
- Global dynamics in an age-structured HIV model with humoral immunity
- Global analysis of a generalized viral infection temporal model with cell-to-cell transmission and absorption effect under therapy
- Threshold dynamics of a general delayed within-host viral infection model with humoral immunity and two modes of virus transmission
- Effects of masks on the transmission of infectious diseases
- Stability and Hopf bifurcation of HIV-1 model with Holling II infection rate and immune delay
- Spatial dynamics of a viral infection model with immune response and nonlinear incidence
- A note on the global properties of an age-structured viral dynamic model with multiple target cell populations
- Local and global stabilities of a viral dynamics model with infection-age and immune response
- An age-structured virus model with two routes of infection in heterogeneous environments
- Threshold asymptotic dynamics for a spatial age-dependent cell-to-cell transmission model with nonlocal disperse
- Global dynamics for an age-structured HIV virus infection model with cellular infection and antiretroviral therapy
- Dynamics of SARS-CoV-2 infection model with two modes of transmission and immune response
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