Oscillatory viral dynamics in a delayed HIV pathogenesis model

DOI10.1016/J.MBS.2009.03.003zbMATH Open1168.92031OpenAlexW2105265365WikidataQ51847325 ScholiaQ51847325MaRDI QIDQ2270535FDOQ2270535

Authors: Yan Wang, Yicang Zhou, Jianhong Wu, Jane M. Heffernan 

Publication date: 28 July 2009

Published in: Mathematical Biosciences (Search for Journal in Brave)

Full work available at URL: https://doi.org/10.1016/j.mbs.2009.03.003

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zbMATH Keywords

HIVantiretroviral therapyasymptotic stabilityHopf bifurcationtime delayT-cells

Mathematics Subject Classification ID

Medical applications (general) (92C50) Medical epidemiology (92C60) Stability theory of functional-differential equations (34K20) Qualitative investigation and simulation of models involving functional-differential equations (34K60) Bifurcation theory of functional-differential equations (34K18)

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Cited In (90)

• Dynamics of two time delays differential equation model to HIV latent infection
• Dynamics analysis and simulation of a modified HIV infection model with a saturated infection rate
• A class of virus dynamic model with inhibitory effect on the growth of uninfected T cells caused by infected T cells and its stability analysis
• Stationary distribution and probability density function analysis of a stochastic HIV model with cell-to-cell infection
• Global stability of delayed viral infection models with nonlinear antibody and CTL immune responses and general incidence rate
• A stochastic HIV infection model with latent infection and antiretroviral therapy
• A stochastic HIV infection model with T-cell proliferation and CTL immune response
• A model of HIV-1 infection with two time delays: mathematical analysis and comparison with patient data
• Modelling a stochastic HIV model with logistic target cell growth and nonlinear immune response function
• Analysis of an HIV infection model with treatments and delayed immune response
• Global dynamics of an in-host viral model with intracellular delay
• Global dynamics of an age-structured within-host viral infection model with cell-to-cell transmission and general humoral immunity response
• Mathematical analysis and clinical implications of an HIV model with adaptive immunity
• Asymptotic behavior of a stochastic delayed HIV-1 infection model with nonlinear incidence
• Analysis of the dynamics of a delayed HIV pathogenesis model
• GLOBAL DYNAMICS IN A MULTI-GROUP EPIDEMIC MODEL FOR DISEASE WITH LATENCY SPREADING AND NONLINEAR TRANSMISSION RATE
• Global dynamics of an HIV infection model with two classes of target cells and distributed delays
• Stationary distribution and extinction of a stochastic viral infection model
• Mathematical analysis of an age-structured HIV model with intracellular delay
• Andronov-Hopf bifurcation and sensitivity analysis of a time-delay HIV model with logistic growth and antiretroviral treatment
• Bifurcation analysis in a model of cytotoxic T-lymphocyte response to viral infections
• Stability analysis in delayed within-host viral dynamics with both viral and cellular infections
• Stability and multi-parametric Hopf bifurcation analyses of viral infection model with time delay
• On global stability of an HIV pathogenesis model with cure rate
• Global dynamics of an age-structured virus model with saturation effects
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• Modeling inhibitory effect on the growth of uninfected T cells caused by infected T cells: stability and Hopf bifurcation
• Impact of intracellular delay, immune activation delay and nonlinear incidence on viral dynamics
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• Dynamic behaviors of a delayed HIV model with stage-structure
• Complete classification of global dynamics of a virus model with immune responses
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• Bifurcation analysis of HIV-1 infection model with cell-to-cell transmission and immune response delay
• Dynamics of cytotoxic T-lymphocytes and helper cells in human immunodeficiency virus infection with Hill-type infection rate and sigmoidal CTL expansion
• Stability and Hopf bifurcation analysis of immune response delayed HIV type 1 infection model with two target cells
• How self-proliferation of CD4\(^{+}\)T cells affect the HIV dynamics in an in-host target-cell limited HIV model with saturation infection rate: a quasi-steady-state approximation analysis
• A delay-dependent model with HIV drug resistance during therapy
• Stochastic model for in-host HIV dynamics with therapeutic intervention
• Global stability for a delayed HIV-1 infection model with nonlinear incidence of infection
• Global dynamics of a spatial heterogeneous viral infection model with intracellular delay and nonlocal diffusion
• Saturation effects for CTL mediated control of HIV-1 infection: a mathematical study
• Global properties of virus dynamics models with multitarget cells and discrete-time delays
• Analysis of an HIV infection model with logistic target-cell growth and cell-to-cell transmission
• The stability analysis of a general viral infection model with distributed delays and multi-staged infected progression
• Global dynamics of a delay virus model with recruitment and saturation effects of immune responses
• Global dynamics for an age-structured HIV virus infection model with cellular infection and antiretroviral therapy
• Analysis and control of an age-structured HIV-1 epidemic model with different transmission mechanisms
• Analysis of a stochastic HIV model with cell-to-cell transmission and Ornstein–Uhlenbeck process
• Viral dynamics of a latent HIV infection model with Beddington-DeAngelis incidence function, B-cell immune response and multiple delays
• Global dynamics of an HIV-1 infection model with distributed intracellular delays
• Modelling heterogeneity in viral-tumour dynamics: the effects of gene-attenuation on viral characteristics
• Existence of order-1 periodic solutions for a viral infection model with state-dependent impulsive control
• A primary infection model for HIV and immune response with two discrete time delays
• Modeling the effect of reactive oxygen species and CTL immune response on HIV dynamics
• Dynamics on Hepatitis B Virus Infection In Vivo with Interval Delay
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• Analysis and optimal control of an HIV model with logistic growth and infected cells in eclipse phase
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• Title not available (Why is that?)
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• Chemotaxis-driven stationary and oscillatory patterns in a diffusive HIV-1 model with CTL immune response and general sensitivity

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