A mathematical model of cell cycle dysregulation due to human papillomavirus infection

DOI10.1007/S11538-017-0299-9MaRDI QIDQ2408853zbMATH OpenOpenAlexWikidataFDO

Authors Anna Miller, Karl Munger, Frederick R. Adler

Publication date 20 October 2017

Published in Bulletin of Mathematical Biology (Search for Journal in Brave)

Full work available at URL http://europepmc.org/articles/pmc5554117

zbMATH Keywords

HPV E7human papillomavirusesmathematical modelretinoblastoma protein

Mathematics Subject Classification ID

Cell biology (92C37) Medical epidemiology (92C60)

Recommendations

• Understanding the mechanisms of HPV-related carcinogenesis: implications for cell cycle dynamics
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• A model of cell cycle behavior dominated by kinetics of a pathway stimulated by growth factors
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Cites work

• A model for restriction point control of the mammalian cell cycle
• Revising ecological assumptions about human papillomavirus interactions and type replacement
• The dynamics of HPV infection and cervical cancer cells

Cited in

(7)

• Understanding the mechanisms of HPV-related carcinogenesis: implications for cell cycle dynamics
• Mathematical analysis of a SIPC age-structured model of cervical cancer
• Mathematical modeling of the cells repair regulations in nasopharyngeal carcinoma
• A cellular mathematical model of nasopharyngeal carcinoma with addition compartment of virus
• A numerical approach for a model of the precancer lesions caused by the human papillomavirus
• A nonlinear transport-diffusion model for the interactions between immune system cells and HPV-infected cells
• Stochastic dynamics of human papillomavirus delineates cervical cancer progression

Describes a project that uses

Uses Software

• diffEq
• deSolve
• RootSolve

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