Axisymmetric solutions for a chemotaxis model of Multiple Sclerosis
DOI10.1007/S11587-018-0406-8zbMATH Open1420.35432OpenAlexW2803691523WikidataQ129769897 ScholiaQ129769897MaRDI QIDQ2425216FDOQ2425216
Authors: Eleonora Bilotta, Valeria Giunta, Pietro Pantano, F. Gargano, M. C. Lombardo, M. Sammartino 
Publication date: 26 June 2019
Published in: Ricerche di Matematica (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1007/s11587-018-0406-8
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Bifurcations in context of PDEs (35B32) Traveling wave solutions (35C07) Reaction-diffusion equations (35K57) PDEs in connection with biology, chemistry and other natural sciences (35Q92) Cell movement (chemotaxis, etc.) (92C17)
Cites Work
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- Demyelination patterns in a mathematical model of multiple sclerosis
- Wavefront invasion for a chemotaxis model of multiple sclerosis
- Title not available (Why is that?)
Cited In (13)
- The effect of chemotaxis on T-cell regulatory dynamics
- Global well-posedness and nonlinear stability of a chemotaxis system modelling multiple sclerosis
- Existence and regularity for a chemotaxis model involved in the modeling of multiple sclerosis
- On the use of a coupled mathematical model for understanding the dynamics of multiple sclerosis
- Demyelination patterns in a mathematical model of multiple sclerosis
- Mathematical description of concentric demyelination in the human brain: self-organization models, from Liesegang rings to chemotaxis
- Subcritical Turing patterns in hyperbolic models with cross-diffusion
- Global asymptotic behavior of solutions for a parabolic-parabolic-ODE chemotaxis system modeling multiple sclerosis
- A minimal unified model of disease trajectories captures hallmarks of multiple sclerosis
- Could mathematics be the key to unlocking the mysteries of multiple sclerosis?
- Pattern formation and transition to chaos in a chemotaxis model of acute inflammation
- Wavefront invasion for a chemotaxis model of multiple sclerosis
- Global boundedness and asymptotic behavior of solutions for a quasilinear chemotaxis model of multiple sclerosis with nonlinear signal secretion
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