Decay bounds in a model for aggregation of microglia: application to Alzheimer's disease senile plaques
DOI10.1098/RSPA.2005.1483zbMATH Open1186.92025OpenAlexW2152324084MaRDI QIDQ5427652FDOQ5427652
Authors: R. A. Quinlan, Brian Straughan
Publication date: 21 November 2007
Published in: Proceedings of the Royal Society A: Mathematical, Physical and Engineering Sciences (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1098/rspa.2005.1483
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Medical applications (general) (92C50) PDEs in connection with biology, chemistry and other natural sciences (35Q92) Cell movement (chemotaxis, etc.) (92C17)
Cites Work
- From 1970 until present: The Keller-Segel model in chemotaxis and its consequences. I
- From 1970 until present: the Keller-Segel model in chemotaxis and its consequences. II
- Chemotactic signaling, microglia, and Alzheimer's disease senile plaques: Is there a connection?
- Exponential attractor for a chemotaxis-growth system of equations
Cited In (15)
- Mathematical modeling of chemotaxis and glial scarring around implanted electrodes
- Chemotactic signaling, microglia, and Alzheimer's disease senile plaques: Is there a connection?
- Decay for a Keller-Segel chemotaxis model
- Global attractor for a chemotaxis model with reaction term
- Global existence for an attraction-repulsion chemotaxis fluid model with logistic source
- Decay estimates in chemotaxis: aggregation of glia and a possible application to Alzheimer's disease senile plaques
- Aggregation of microglia in 2D with string gradient weighted moving finite elements
- Global existence and blow-up for a system describing the aggregation of microglia
- A qualitative model for aggregation and diffusion of \(\beta \)-amyloid in Alzheimer's disease
- Demyelination patterns in a mathematical model of multiple sclerosis
- Axisymmetric solutions for a chemotaxis model of Multiple Sclerosis
- Global existence for an attraction-repulsion chemotaxis-fluid system in a framework of Besov-Morrey type
- A dual-gradient chemotaxis system modeling the spontaneous aggregation of microglia in Alzheimer’s disease
- Wavefront invasion for a chemotaxis model of multiple sclerosis
- Analysis and numerical simulations of a chemotaxis model of aggregation of microglia in Alzheimer's disease
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