An intracellular \(\text{Ca}^{2+}\) subsystem as a biologically plausible source of intrinsic conditional bistability in a network model of working memory
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Publication:2500141
DOI10.1007/s10827-006-4791-8zbMath1119.92010OpenAlexW2013350381WikidataQ46971562 ScholiaQ46971562MaRDI QIDQ2500141
Christopher P. Fall, John Rinzel
Publication date: 23 August 2006
Published in: Journal of Computational Neuroscience (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1007/s10827-006-4791-8
computational modelcalcium signalingworking memoryneuromodulatorspersistent activityinositol 1,4,5 trisphosphate
Related Items (3)
Neuronal Calcium Wave Propagation Varies with Changes in Endoplasmic Reticulum Parameters: A Computer Model ⋮ Three generic bistable scenarios of the interplay of voltage pulses and gene expression in neurons ⋮ Neural energy mechanism and neurodynamics of memory transformation
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Cites Work
- Dynamics of pattern formation in lateral-inhibition type neural fields
- A model of visuospatial working memory in prefrontal cortex: Recurrent network and cellular bistability
- Background-activity-dependent properties of a network model for working memory that incorporates cellular bistability
- A mathematical theory of the functional dynamics of cortical and thalamic nervous tissue
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