Mathematical modelling of tissue invasion
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Publication:2809031
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- Numerical solutions for a model of tissue invasion and migration of tumour cells
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- Some further progress for boundedness of solutions to a quasilinear higher-dimensional chemotaxis-haptotaxis model with nonlinear diffusion
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- A density-dependent chemotaxis-haptotaxis system modeling cancer invasion
- Global dynamics of a quasilinear chemotaxis model arising from tumor invasion
- Global boundedness for a \(N\)-dimensional two species cancer invasion haptotaxis model with tissue remodeling
- Slow and fast invasion waves in a model of acid-mediated tumour growth
- Numerical versus experimental data for prostate tumour growth
- Global asymptotic stability in a chemotaxis-growth model for tumor invasion
- Effects of density-suppressed motility in a two-dimensional chemotaxis model arising from tumor invasion
- Local existence and uniqueness of regular solutions in a model of tissue invasion by solid tumours
- Local existence and uniqueness of solutions to approximate systems of 1D tumor invasion model
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- Singular structure formation in a degenerate haptotaxis model involving myopic diffusion
- Adaptive grid modelling for cancer cells in the early stage of invasion
- Positivity-preserving nonstandard finite difference schemes for cross-diffusion equations in biosciences
- MICRO AND MESO SCALES OF DESCRIPTION CORRESPONDING TO A MODEL OF TISSUE INVASION BY SOLID TUMOURS
- Numerical analysis of a chemotaxis model for tumor invasion
- Propagation of chaos in the nonlocal adhesion models for two cancer cell phenotypes
- Existence of solutions and optimal control for a model of tissue invasion by solid tumours
- Quasi-variational structure approach to systems with degenerate diffusions
- Nonlocal adhesion models for two cancer cell phenotypes in a multidimensional bounded domain
- Global weak solutions to an oncolytic viral therapy model with doubly haptotactic terms
- Global existence for a haptotaxis model of cancer invasion with tissue remodeling
- Boundedness and stabilization of a chemotaxis model describing tumor invasion with signal-dependent motility
- Existence of solutions and local null controllability for a model of tissue invasion by solid tumors
- The existence of solutions for tumor invasion models with time and space dependent diffusion
- Stabilization in a chemotaxis model for tumor invasion
- A Review of Vasculogenesis Models
- Mathematical research for models which is related to chemotaxis system
- Global solutions and asymptotic behavior for a parabolic degenerate coupled system arising from biology
- A note for global existence of a two-dimensional chemotaxis-haptotaxis model with remodeling of non-diffusible attractant
- Asymptotic behavior of global solutions to a model of cell invasion
- Boundedness and large time behavior in a quasilinear chemotaxis model for tumor invasion
- Convergence and positivity of finite element methods for a haptotaxis model of tumoral invasion
- Global boundedness of classical solutions to a two species cancer invasion haptotaxis model with tissue remodeling
- Boundedness of solutions to a quasilinear chemotaxis-haptotaxis model
- Mathematical modeling of invadopodia formation
- Boundedness in a quasilinear chemotaxis-haptotaxis system with logistic source
- Global boundedness in a three-dimensional chemotaxis-haptotaxis model
- Large time behavior in a chemotaxis model with nonlinear general diffusion for tumor invasion
- Boundedness of solutions to a quasilinear higher-dimensional chemotaxis-haptotaxis model with nonlinear diffusion
- MATHEMATICAL MODELLING OF CANCER INVASION OF TISSUE: THE ROLE AND EFFECT OF NONLOCAL INTERACTIONS
- Boundedness and large-time behavior in a chemotaxis system with signal-dependent motility arising from tumor invasion
- Non-linear evolution equations with non-local coefficients and zero-Neumann condition: one dimensional case
- Selected aspects of avascular tumor growth reproduced by a hybrid model of cell dynamics and chemical kinetics
- Global asymptotic stability in a two-dimensional chemotaxis model arising from tumor invasion
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