What can trigger the onset of Parkinson's disease -- A modeling study based on a compartmental model of -synuclein transport and aggregation in neurons
From MaRDI portal
Publication:309989
DOI10.1016/J.MBS.2016.05.002zbMATH Open1346.92018OpenAlexW2394863193WikidataQ40664492 ScholiaQ40664492MaRDI QIDQ309989FDOQ309989
Authors: I. A. Kuznetsov, A. V. Kuznetsov
Publication date: 7 September 2016
Published in: Mathematical Biosciences (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.1016/j.mbs.2016.05.002
Recommendations
- Can the lack of fibrillar form of alpha-synuclein in Lewy bodies be explained by its catalytic activity?
- Simulating the effect of formation of amyloid plaques on aggregation of tau protein
- Alzheimer's disease: a mathematical model for onset and progression
- How the formation of amyloid plaques and neurofibrillary tangles may be related: a mathematical modelling study
- BLENX MODELS OF α-SYNUCLEIN AND PARKIN KINETICS IN NEUROPATHOLOGY OF PARKINSON'S DISEASE
Cited In (4)
- Can the lack of fibrillar form of alpha-synuclein in Lewy bodies be explained by its catalytic activity?
- Fractionally delineate the neuroprotective function of calbindin-D28k in Parkinson’s disease
- BLENX MODELS OF α-SYNUCLEIN AND PARKIN KINETICS IN NEUROPATHOLOGY OF PARKINSON'S DISEASE
- Modelling transport and mean age of dense core vesicles in large axonal arbours
This page was built for publication: What can trigger the onset of Parkinson's disease -- A modeling study based on a compartmental model of \(\alpha\)-synuclein transport and aggregation in neurons
Report a bug (only for logged in users!)Click here to report a bug for this page (MaRDI item Q309989)