A partial differential equation model of metastasized prostatic cancer
DOI10.3934/MBE.2013.10.591zbMATH Open1268.92059OpenAlexW2334453129WikidataQ43636059 ScholiaQ43636059MaRDI QIDQ356925FDOQ356925
Authors: A. Friedman, Harsh Vardhan Jain
Publication date: 29 July 2013
Published in: Mathematical Biosciences and Engineering (Search for Journal in Brave)
Full work available at URL: https://doi.org/10.3934/mbe.2013.10.591
Recommendations
- Modeling prostate cancer response to continuous versus intermittent androgen ablation therapy
- Development of a mathematical model that predicts the outcome of hormone therapy for prostate cancer
- A partial differential equation model and its reduction to an ordinary differential equation model for prostate tumor growth under intermittent hormone therapy
- Mathematical analysis and simulation study of a phase-field model of prostate cancer growth with chemotherapy and antiangiogenic therapy effects
- A mathematical model of prostate tumor growth under hormone therapy with mutation inhibitor
Medical applications (general) (92C50) PDEs in connection with biology, chemistry and other natural sciences (35Q92) Free boundary problems for PDEs (35R35)
Cited In (11)
- Correlation between animal and mathematical models for prostate cancer progression
- Long time dynamics of a phase-field model of prostate cancer growth with chemotherapy and antiangiogenic therapy effects
- A numerical simulation study of the dual role of \(5\alpha\)-reductase inhibitors on tumor growth in prostates enlarged by benign prostatic hyperplasia via stress relaxation and apoptosis upregulation
- An alternative approach to modelling relapse in cancer with an application to adenocarcinoma of the prostate
- Mathematical insights into neuroendocrine transdifferentiation of human prostate cancer cells
- An ordinary differential equation model for the multistep transformation to cancer
- Mathematical analysis and simulation study of a phase-field model of prostate cancer growth with chemotherapy and antiangiogenic therapy effects
- Solitary solutions to an androgen-deprivation prostate cancer treatment model
- Modeling prostate cancer response to continuous versus intermittent androgen ablation therapy
- Maximum bound principle and non-negativity preserving ETD schemes for a phase field model of prostate cancer growth with treatment
- An integrative model of prostate cancer interaction with the bone microenvironment
This page was built for publication: A partial differential equation model of metastasized prostatic cancer
Report a bug (only for logged in users!)Click here to report a bug for this page (MaRDI item Q356925)